Suppression of CED-3-independent apoptosis by mitochondrial betaNAC in Caenorhabditis elegans. - Wikidata - awgldk - TriplyDB

Suppression of CED-3-independent apoptosis by mitochondrial betaNAC in Caenorhabditis elegans.

Suppression of CED-3-independent apoptosis by mitochondrial betaNAC in Caenorhabditis elegans.

http://www.wikidata.org/entity/Q45973814

about

C. elegans EIF-3.K promotes programmed cell death through CED-3 caspase Depletion of the C. elegans NAC engages the unfolded protein response, resulting in increased chaperone expression and apoptosis.L25 functions as a conserved ribosomal docking site shared by nascent chain-associated complex and signal-recognition particle.Mechanism of small heat shock protein function in vivo: a knock-in mouse model demonstrates that the R49C mutation in alpha A-crystallin enhances protein insolubility and cell death Neuron-specific knock-down of SMN1 causes neuron degeneration and death through an apoptotic mechanism Hypoxic preconditioning requires the apoptosis protein CED-4 in C. elegans.Coenzyme Q protects Caenorhabditis elegans GABA neurons from calcium-dependent degeneration.Postembryonic RNAi in Heterorhabditis bacteriophora: a nematode insect parasite and host for insect pathogenic symbionts.Bicaudal is a conserved substrate for Drosophila and mammalian caspases and is essential for cell survival A Caenorhabditis elegans model of insulin resistance: altered macronutrient storage and dauer formation in an OGT-1 knockout.Defining the specificity of cotranslationally acting chaperones by systematic analysis of mRNAs associated with ribosome-nascent chain complexes.Noncanonical cell death in the nematode Caenorhabditis elegans.Dual binding mode of the nascent polypeptide-associated complex reveals a novel universal adapter site on the ribosome.The nascent polypeptide-associated complex is a key regulator of proteostasis.The nascent polypeptide-associated complex is essential for autophagic flux.Restriction of vaccinia virus replication by a ced-3 and ced-4-dependent pathway in Caenorhabditis elegans Human beta casein fragment (54-59) modulates M. bovis BCG survival and basic transcription factor 3 (BTF3) expression in THP-1 cell line.A conserved motif is prerequisite for the interaction of NAC with ribosomal protein L23 and nascent chains.Inhibition of a basal transcription factor 3-like gene Osj10gBTF3 in rice results in significant plant miniaturization and typical pollen abortion.ced-4 and proto-oncogene tfg-1 antagonistically regulate cell size and apoptosis in C. elegans Functional Dissection of the Nascent Polypeptide-Associated Complex in Saccharomyces cerevisiae Ribosome association and stability of the nascent polypeptide-associated complex is dependent upon its own ubiquitination.Noncanonical cell death programs in the nematode Caenorhabditis elegans.Cell-nonautonomous inhibition of radiation-induced apoptosis by dynein light chain 1 in Caenorhabditis elegans Adenine nucleotide translocator cooperates with core cell death machinery to promote apoptosis in Caenorhabditis elegans.Cell Death in C. elegans Development.RNA aptamers targeting the cell death inhibitor CED-9 induce cell killing in Caenorhabditis elegans.Exploring the roles of basal transcription factor 3 in eukaryotic growth and development.GSK-3β-dependent downregulation of γ-taxilin and αNAC merge to regulate ER stress responses Silencing of TaBTF3 gene impairs tolerance to freezing and drought stresses in wheat.Quantitative proteomic analysis of induced pluripotent stem cells derived from a human Huntington's disease patient.alphaNAC depletion as an initiator of ER stress-induced apoptosis in hypoxia.Programmed Cell Death During Caenorhabditis elegans Development Caenorhabditis elegans drp-1 and fis-2 regulate distinct cell-death execution pathways downstream of ced-3 and independent of ced-9.The yeast ubiquitin ligase Rsp5 downregulates the alpha subunit of nascent polypeptide-associated complex Egd2 under stress conditions.Transcriptional control of non-apoptotic developmental cell death in C. elegans.αβ'-NAC cooperates with Sam37 to mediate early stages of mitochondrial protein import.The crystal structure of archaeal nascent polypeptide-associated complex (NAC) reveals a unique fold and the presence of a ubiquitin-associated domain.

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P2860

Suppression of CED-3-independent apoptosis by mitochondrial betaNAC in Caenorhabditis elegans.

http://www.wikidata.org/entity/Q45973814

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

@wuu

2003年学术文章

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2003年学术文章

@zh-hans

2003年学术文章

@zh-my

2003年学术文章

@zh-sg

2003年學術文章

@yue

2003年學術文章

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2003年學術文章

@zh-hant

name

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@en

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@nl

type

label

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@en

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@nl

prefLabel

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@en

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@nl

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P1476

Suppression of CED-3-independe ...... NAC in Caenorhabditis elegans.

@en

P2093

Eric S Witze

http://www.w3.org/2001/XMLSchema#string

Joel H Rothman

http://www.w3.org/2001/XMLSchema#string

Tim A Bloss

http://www.w3.org/2001/XMLSchema#string

P2860

Proteolytic activation of PKN by caspase-3 or related protease during apoptosis

Crystal structure of Apaf-1 caspase recruitment domain: an alpha-helical Greek key fold for apoptotic signaling

Nascent-polypeptide-associated complex

The embryonic cell lineage of the nematode Caenorhabditis elegans

Caenorhabditis elegans gene ced-9 protects cells from programmed cell death

Large-scale analysis of gene function in Caenorhabditis elegans by high-throughput RNAi

Preparative high-resolution two-dimensional electrophoresis enables the identification of RNA polymerase B transcription factor 3 as an apoptosis-associated protein in the human BL60-2 Burkitt lymphoma cell line.

Predominant identification of RNA-binding proteins in Fas-induced apoptosis by proteome analysis.

The molecular mechanism of programmed cell death in C. elegans.

The Caenorhabditis elegans mucolipin-like gene cup-5 is essential for viability and regulates lysosomes in multiple cell types.

P2888

P304

1066-1071

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scholarly article

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10.1038/NATURE01920

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P433

6952

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P478

424

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2003-08-01T00:00:00Z

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10592172

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1000655081

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12944970

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P921

Caenorhabditis elegans