N-glycan processing deficiency promotes spontaneous inflammatory demyelination and neurodegeneration. - Wikidata - awgldk - TriplyDB

N-glycan processing deficiency promotes spontaneous inflammatory demyelination and neurodegeneration.

N-glycan processing deficiency promotes spontaneous inflammatory demyelination and neurodegeneration.

http://www.wikidata.org/entity/Q45994343

about

N-Acetylglucosamine Inhibits T-helper 1 (Th1)/T-helper 17 (Th17) Cell Responses and Treats Experimental Autoimmune Encephalomyelitis Roles for major histocompatibility complex glycosylation in immune function Encoding asymmetry of the N-glycosylation motif facilitates glycoprotein evolution Protein kinase C θ regulates the phenotype of murine CD4+ Th17 cells Glycolysis and glutaminolysis cooperatively control T cell function by limiting metabolite supply to N-glycosylation.Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis.Sugar-free frosting, a homolog of SAD kinase, drives neural-specific glycan expression in the Drosophila embryo.Galectin-glycan lattices regulate cell-surface glycoprotein organization and signalling.Increased sensitivity of Apolipoprotein E knockout mice to swainsonine dependent immunomodulation.Family studies of type 1 diabetes reveal additive and epistatic effects between MGAT1 and three other polymorphisms Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: an update for 2007-2008.CD40 glycoforms and TNF-receptors 1 and 2 in the formation of CD40 receptor(s) in autoimmunity Galectin-9 controls CD40 signaling through a Tim-3 independent mechanism and redirects the cytokine profile of pathogenic T cells in autoimmunity.T-cell growth, cell surface organization, and the galectin-glycoprotein lattice.Genetics and the environment converge to dysregulate N-glycosylation in multiple sclerosis.N-Glycans differentially regulate eosinophil and neutrophil recruitment during allergic airway inflammation Mgat5 deficiency in T cells and experimental autoimmune encephalomyelitis.Interleukin-2, Interleukin-7, T cell-mediated autoimmunity, and N-glycosylation.Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants.Glucosamine Modulates T Cell Differentiation through Down-regulating N-Linked Glycosylation of CD25.The regulatory power of glycans and their binding partners in immunity Glycosylation and other PTMs alterations in neurodegenerative diseases: Current status and future role in neurotrauma.Mammalian glycosylation in immunity.Role of endothelial N-glycan mannose residues in monocyte recruitment during atherogenesis.Inhibition of calpain attenuates encephalitogenicity of MBP-specific T cells.T cell receptor signaling co-regulates multiple Golgi genes to enhance N-glycan branching.Pathogenesis of multiple sclerosis via environmental and genetic dysregulation of N-glycosylation.Metabolism, cell surface organization, and disease.The genetics of multiple sclerosis: an up-to-date review.Copy number variation in pediatric multiple sclerosis.Myelin oligodendrocyte glycoprotein has a dual role in T cell autoimmunity against central nervous system myelin.Abnormal galactosylation of immunoglobulin G in cerebrospinal fluid of multiple sclerosis patients.Distinctive Surface Glycosylation Patterns Associated With Mouse and Human CD4+ Regulatory T Cells and Their Suppressive Function.N-glycosylation bidirectionally extends the boundaries of thymocyte positive selection by decoupling Lck from Ca²⁺ signaling.Neurophagy, the phagocytosis of live neurons and synapses by glia, contributes to brain development and disease.The Modulatory Roles of N-glycans in T-Cell-Mediated Autoimmune Diseases.Galectin-1 enhances TNFα-induced inflammatory responses in Sertoli cells through activation of MAPK signalling.

P2860

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P2860

N-glycan processing deficiency promotes spontaneous inflammatory demyelination and neurodegeneration.

http://www.wikidata.org/entity/Q45994343

description

2007 nî lūn-bûn

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2007年の論文

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2007年學術文章

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2007年學術文章

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name

N-glycan processing deficiency ...... ination and neurodegeneration.

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N-glycan processing deficiency ...... ination and neurodegeneration.

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type

label

N-glycan processing deficiency ...... ination and neurodegeneration.

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N-glycan processing deficiency ...... ination and neurodegeneration.

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prefLabel

N-glycan processing deficiency ...... ination and neurodegeneration.

@en

N-glycan processing deficiency ...... ination and neurodegeneration.

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Journal of Biological Chemistry

P1476

N-glycan processing deficiency ...... ination and neurodegeneration.

@en

P2093

Ani Grigorian

http://www.w3.org/2001/XMLSchema#string

Guoyan Gao

http://www.w3.org/2001/XMLSchema#string

I-Ju Chen

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Judy Pawling

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Michael Demetriou

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Sung-Uk Lee

http://www.w3.org/2001/XMLSchema#string

Tahseen Mozaffar

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P2860

Gray matter involvement in multiple sclerosis

Identification of a lectin causing the degeneration of neuronal processes using engineered embryonic stem cells

A randomized, placebo-controlled trial of natalizumab for relapsing multiple sclerosis

Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation.

Suppression of tumor growth and metastasis in Mgat5-deficient mice.

Phagocytosis and clearance of apoptotic cells is mediated by MER.

Genetic models for CNS inflammation.

T-cell regulation by CD28 and CTLA-4.

Oligosaccharide specificity of galectins: a search by frontal affinity chromatography.

Fulminant spontaneous autoimmunity of the central nervous system in mice transgenic for the myelin proteolipid protein-specific T cell receptor.

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10.1074/JBC.M704839200

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46

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282

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