Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

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Changes in cell junctions induced by inhibition of epidermal growth factor receptor in oral squamous cell carcinoma cells

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Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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name

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

@en

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

@nl

type

Item

label

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

@en

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

@nl

prefLabel

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

@en

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

@nl

P1476

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer

@en

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F T Salles

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K U Sales

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R M Castilho

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Molecular cloning of the akt oncogene and its human homologues AKT1 and AKT2: amplification of AKT1 in a primary human gastric adenocarcinoma

Intercellular adhesion, signalling and the cytoskeleton

Inhibition of cyclin D1 phosphorylation on threonine-286 prevents its rapid degradation via the ubiquitin-proteasome pathway

Glycogen synthase kinase-3beta regulates cyclin D1 proteolysis and subcellular localization

The phosphatidylinositol 3-Kinase AKT pathway in human cancer

The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase

Beta-catenin mediates the interaction of the cadherin-catenin complex with epidermal growth factor receptor.

Cadherin-bound beta-catenin feeds into the Wnt pathway upon adherens junctions dissociation: evidence for an intersection between beta-catenin pools.

Effect of phosphoinositide-dependent kinase 1 on protein kinase B translocation and its subsequent activation.

A possible role for the WNT-1 pathway in oral carcinogenesis.

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e42-8

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scholarly article

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10.1111/ODI.12124

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Décio Pinto

Aline Corrêa Abrahão

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2013-06-04T00:00:00Z

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Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

about

P2860

P2860

Cyclin D1-induced proliferation is independent of beta-catenin in head and neck cancer.

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P2860

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P698