Over-expression of forkhead box P3 and its association with receptor activator of nuclear factor-kappa B ligand, interleukin (IL) -17, IL-10 and transforming growth factor-beta during the progression of chronic periodontitis.
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Host response mechanisms in periodontal diseasesSuppression of αvβ6 Integrin Expression by Polymicrobial Oral Biofilms in Gingival Epithelial CellsIL-17 in sera from patients with aggressive periodontitisBasic biology and role of interleukin-17 in immunity and inflammationThe Influence of Interleukin 17A and IL17F Polymorphisms on Chronic Periodontitis Disease in Brazilian Patients.Periodontitis increases rheumatic factor serum levels and citrullinated proteins in gingival tissues and alter cytokine balance in arthritic rats.Association between polymorphisms in interleukin-17A and -17F genes and chronic periodontal disease.Serum, saliva, and GCF concentration of RANKL and osteoprotegerin in smokers versus nonsmokers with chronic periodontitisPrevention of inflammation-mediated bone loss in murine and canine periodontal disease via recruitment of regulatory lymphocytes.Expression profile of IL-35 mRNA in gingiva of chronic periodontitis and aggressive periodontitis patients: a semiquantitative RT-PCR study.Complement and periodontitis.How has research into cytokine interactions and their role in driving immune responses impacted our understanding of periodontitis?Host-derived diagnostic markers related to soft tissue destruction and bone degradation in periodontitis.Epithelial integrins with special reference to oral epithelia.Host-pathogen interactions in progressive chronic periodontitis.T-helper cells in the etiopathogenesis of periodontal disease: A mini review.The RANKL-OPG system in clinical periodontology.Inflammatory and immune pathways in the pathogenesis of periodontal disease.The presence, function and regulation of IL-17 and Th17 cells in periodontitis.Altered gene expression in gingival tissues and enhanced bone loss in rats with diabetes with experimental periodontitis.Forkhead box P3-positive regulatory T-cells and interleukin 17-positive T-helper 17 cells in chronic inflammatory periodontal disease.T- and B-cell subsets in periodontitis.Local inflammatory reactions in patients with diabetes and periodontitis.Denervation effectively aggravates rat experimental periodontitis.IL-17: overview and role in oral immunity and microbiome.Activation of RANKL-induced osteoclasts and memory T lymphocytes by Porphyromonas gingivalis is serotype dependant.Resveratrol decreases periodontal breakdown and modulates local levels of cytokines during periodontitis in rats.B10 Cells Alleviate Periodontal Bone Loss in Experimental Periodontitis.Expression levels of th-2 and th-17 characteristic genes in healthy tissue versus periodontitis.Variability of the dendritic cell response triggered by different serotypes of Aggregatibacter actinomycetemcomitans or Porphyromonas gingivalis is toll-like receptor 2 (TLR2) or TLR4 dependent.Evaluation of transcription factor that regulates T helper 17 and regulatory T cells function in periodontal health and disease.Increased expression of interleukin (IL)-35 and IL-17, but not IL-27, in gingival tissues with chronic periodontitis.Angiopoietin-like protein 2 regulates Porphyromonas gingivalis lipopolysaccharide-induced inflammatory response in human gingival epithelial cells.IL-23-producing CD68(+) macrophage-like cells predominate within an IL-17-polarized infiltrate in chronic periodontitis lesions.Oral administration of all-trans retinoic acid suppresses experimental periodontitis by modulating the Th17/Treg imbalance.The function of dendritic cells in modulating the host response.A preliminary study on the FAM5C expression in generalized chronic periodontitis.IL-35 may maintain homeostasis of the immune microenvironment in periodontitis.Chronic Periodontitis and RANKL/OPG Ratio in Peri-Implant Mucosae Inflammation.Human periodontal ligament fibroblasts synthesize C-reactive protein and Th-related cytokines in response to interleukin (IL)-6 trans-signalling.
P2860
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P2860
Over-expression of forkhead box P3 and its association with receptor activator of nuclear factor-kappa B ligand, interleukin (IL) -17, IL-10 and transforming growth factor-beta during the progression of chronic periodontitis.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@en
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@nl
type
label
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@en
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@nl
prefLabel
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@en
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@nl
P2093
P1476
Over-expression of forkhead bo ...... sion of chronic periodontitis.
@en
P2093
Augusto Silva
Jorge Gamonal
Mariano Sanz
Nicolás Dutzan
Rolando Vernal
P304
P356
10.1111/J.1600-051X.2009.01390.X
P407
P577
2009-05-01T00:00:00Z