Contribution of fluid shear response in leukocytes to hemodynamic resistance in the spontaneously hypertensive rat.
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Autonomic-Immune-Vascular Interaction: An Emerging Concept for Neurogenic HypertensionApplications of computational models to better understand microvascular remodelling: a focus on biomechanical integration across scales.Spontaneous white matter damage, cognitive decline and neuroinflammation in middle-aged hypertensive rats: an animal model of early-stage cerebral small vessel diseaseDanhong injection reduces vascular remodeling and up-regulates the Kallikrein-kinin system in spontaneously hypertensive rats.Leukocyte membrane bleb and pseudopod formation in hypertensionReceptor cleavage reduces the fluid shear response in neutrophils of the spontaneously hypertensive ratMembrane cholesterol modulates the fluid shear stress response of polymorphonuclear leukocytes via its effects on membrane fluidityAngiogenesis in mesenteric microvascular networks from spontaneously hypertensive versus normotensive rats.Receptor cleavage and P-selectin-dependent reduction of leukocyte adhesion in the spontaneously hypertensive rat.The effect of microvascular pattern alterations on network resistance in spontaneously hypertensive rats.Proteolytic receptor cleavage in the pathogenesis of blood rheology and co-morbidities in metabolic syndrome. Early forms of autodigestion.An emerging role of degrading proteinases in hypertension and the metabolic syndrome: autodigestion and receptor cleavage.Pravastatin improves postprandial endothelial dysfunction and hemorheological deterioration in patients with effort angina pectoris.THE AUTODIGESTION HYPOTHESIS AND RECEPTOR CLEAVAGE IN DIABETES AND HYPERTENSION.Dihydropyridine calcium channel blockers inhibit non-esterified-fatty-acid-induced endothelial and rheological dysfunction.Control of oxidative stress in microcirculation of spontaneously hypertensive rats.The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.Excessive leukotriene B4 in nucleus tractus solitarii is prohypertensive in spontaneously hypertensive rats.Chapter 12. Structure of microvascular networks in genetic hypertension.Control of neutrophil pseudopods by fluid shear: role of Rho family GTPases.Characterization of the shear stress regulation of CD18 surface expression by HL60-derived neutrophil-like cells.Fluid shear-induced cathepsin B release in the control of Mac1-dependent neutrophil adhesion.Blood-brain barrier dysfunction and recovery after ischemic stroke.Go with the flow: GEF-H1 mediated shear stress mechanotransduction in neutrophils.Elevated plasma level of soluble F11 receptor/junctional adhesion molecule-A (F11R/JAM-A) in hypertension.Quantitative Indexes of Leukocytes in Spontaneously Hypertensive Rats During Various Periods of Arterial Hypertension Development.Biomechanical aspects of the auto-digestion theory.
P2860
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P2860
Contribution of fluid shear response in leukocytes to hemodynamic resistance in the spontaneously hypertensive rat.
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2004 nî lūn-bûn
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name
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@en
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@nl
type
label
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@en
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@nl
prefLabel
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@en
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@nl
P2093
P1433
P1476
Contribution of fluid shear re ...... pontaneously hypertensive rat.
@en
P2093
Geert W Schmid-Schönbein
Nahoko Ikeda
Nobuhiko Kobayashi
Shunichi Fukuda
Takanori Yasu
P304
P356
10.1161/01.RES.0000133677.77465.38
P577
2004-05-27T00:00:00Z