Regulation of antisense RNA expression during cardiac MHC gene switching in response to pressure overload.
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Hypothesis: Artifacts, Including Spurious Chimeric RNAs with a Short Homologous Sequence, Caused by Consecutive Reverse Transcriptions and Endogenous Random PrimersThe primary microRNA-208b interacts with Polycomb-group protein, Ezh2, to regulate gene expression in the heartMammalian target of rapamycin is essential for cardiomyocyte survival and heart development in miceRegulation of an antisense RNA with the transition of neonatal to IIb myosin heavy chain during postnatal development and hypothyroidism in rat skeletal musclePotential pitfalls in the accuracy of analysis of natural sense-antisense RNA pairs by reverse transcription-PCRIt Is Imperative to Establish a Pellucid Definition of Chimeric RNA and to Clear Up a Lot of Confusion in the Relevant Research.Differential regulation of the myosin heavy chain genes alpha and beta in rat atria and ventricles: role of antisense RNADevelopment of dilated cardiomyopathy in Bmal1-deficient miceHDAC inhibition attenuates cardiac hypertrophy by acetylation and deacetylation of target genes.Intergenic bidirectional promoter and cooperative regulation of the IIx and IIb MHC genes in fast skeletal muscle.Factors controlling cardiac myosin-isoform shift during hypertrophy and heart failuremiR-155 functions downstream of angiotensin II receptor subtype 1 and calcineurin to regulate cardiac hypertrophy.Increase in cardiac myosin heavy-chain (MyHC) alpha protein isoform in hibernating ground squirrels, with echocardiographic visualization of ventricular wall hypertrophy and prolonged contractionCalcineurin plays a modulatory role in loading-induced regulation of type I myosin heavy chain gene expression in slow skeletal muscle.Interplay of chromatin modifications and non-coding RNAs in the heartNon-coding RNAs in homeostasis, disease and stress responses: an evolutionary perspective.Characterization and regulation of mechanical loading-induced compensatory muscle hypertrophy.Chromatin modifications remodel cardiac gene expression.Distribution of histone3 lysine 4 trimethylation at T3-responsive loci in the heart during reversible changes in gene expression.Reversible epigenetic modifications of the two cardiac myosin heavy chain genes during changes in expression.The H19 long noncoding RNA is a novel negative regulator of cardiomyocyte hypertrophy.Transcriptome profiling using single-molecule direct RNA sequencing.The Function and Therapeutic Potential of Long Non-coding RNAs in Cardiovascular Development and Disease.Activity of the beta-myosin heavy chain antisense promoter responds to diabetes and hypothyroidism.Cardiac MHC gene expression: more complexity and a step forward.Hypoxia favors myosin heavy chain beta gene expression in an Hif-1alpha-dependent manner.Capturing the 'ome': the expanding molecular toolbox for RNA and DNA library construction.Current Advances in Noncoding RNA Relevant to Epigenetic Mechanisms
P2860
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P2860
Regulation of antisense RNA expression during cardiac MHC gene switching in response to pressure overload.
description
2006 nî lūn-bûn
@nan
2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
Regulation of antisense RNA ex ...... response to pressure overload.
@en
Regulation of antisense RNA ex ...... response to pressure overload.
@nl
type
label
Regulation of antisense RNA ex ...... response to pressure overload.
@en
Regulation of antisense RNA ex ...... response to pressure overload.
@nl
prefLabel
Regulation of antisense RNA ex ...... response to pressure overload.
@en
Regulation of antisense RNA ex ...... response to pressure overload.
@nl
P2093
P2860
P1476
Regulation of antisense RNA ex ...... response to pressure overload.
@en
P2093
Baldwin KM
P2860
P304
P356
10.1152/AJPHEART.01111.2005
P577
2006-01-13T00:00:00Z