c-Jun N-terminal kinase regulates mitochondrial bioenergetics by modulating pyruvate dehydrogenase activity in primary cortical neurons.
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Induction of glucose metabolism in stimulated T lymphocytes is regulated by mitogen-activated protein kinase signalingRegulation of mitochondrial functions by protein phosphorylation and dephosphorylationSchisandrin B as a hormetic agent for preventing age-related neurodegenerative diseasesRedox regulation of mitochondrial functionPre-symptomatic activation of antioxidant responses and alterations in glucose and pyruvate metabolism in Niemann-Pick Type C1-deficient murine brainLipoic acid restores age-associated impairment of brain energy metabolism through the modulation of Akt/JNK signaling and PGC1α transcriptional pathwayc-Jun N-terminal kinase (JNK)-dependent acute liver injury from acetaminophen or tumor necrosis factor (TNF) requires mitochondrial Sab protein expression in miceRecombinant human mitochondrial transcription factor A stimulates mitochondrial biogenesis and ATP synthesis, improves motor function after MPTP, reduces oxidative stress and increases survival after endotoxinRedox regulation of tumor necrosis factor signalingSilencing glycogen synthase kinase-3beta inhibits acetaminophen hepatotoxicity and attenuates JNK activation and loss of glutamate cysteine ligase and myeloid cell leukemia sequence 1.Visualization of JNK activity dynamics with a genetically encoded fluorescent biosensor.Minimal peroxide exposure of neuronal cells induces multifaceted adaptive responsesERK/MAPK is essential for endogenous neuroprotection in SCN2.2 cellsMitochondrial base excision repair assaysEnergy crisis: the role of oxidative phosphorylation in acute inflammation and sepsisCrosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectivesMouse Models of Oxidative Stress Indicate a Role for Modulating Healthy Aging.Role of nitric oxide-mediated glutathionylation in neuronal function: potential regulation of energy utilizationMitochondrial c-Jun N-terminal kinase (JNK) signaling initiates physiological changes resulting in amplification of reactive oxygen species generation.Selective inhibition of mitochondrial JNK signaling achieved using peptide mimicry of the Sab kinase interacting motif-1 (KIM1).Silencing of nicotinamide nucleotide transhydrogenase impairs cellular redox homeostasis and energy metabolism in PC12 cells.Oxidized low-density lipoprotein-activated c-Jun NH2-terminal kinase regulates manganese superoxide dismutase ubiquitination: implication for mitochondrial redox status and apoptosis.Blocking c-Jun N-terminal kinase (JNK) translocation to the mitochondria prevents 6-hydroxydopamine-induced toxicity in vitro and in vivo.Inhibition of JNK mitochondrial localization and signaling is protective against ischemia/reperfusion injury in rats.Regulation of H(2)O(2)-induced necrosis by PKC and AMP-activated kinase signaling in primary cultured hepatocytes.Role of cellular bioenergetics in smooth muscle cell proliferation induced by platelet-derived growth factor.Neuronal inputs and outputs of aging and longevityActivation of c-Jun-N-terminal kinase and decline of mitochondrial pyruvate dehydrogenase activity during brain aging.Kinetics of the translocation and phosphorylation of alphaB-crystallin in mouse heart mitochondria during ex vivo ischemiaDecreased in vitro mitochondrial function is associated with enhanced brain metabolism, blood flow, and memory in Surf1-deficient mice.In vivo genetic dissection of tumor growth and the Warburg effectEffects and mechanisms of chinese herbal medicine in ameliorating myocardial ischemia-reperfusion injury.Mitochondrial kinases in Parkinson's disease: converging insights from neurotoxin and genetic modelsMitochondrial energy metabolism and redox signaling in brain aging and neurodegenerationCell biology of ischemia/reperfusion injury.The energy-redox axis in aging and age-related neurodegeneration.Lipoic acid: energy metabolism and redox regulation of transcription and cell signalingRole of JNK translocation to mitochondria leading to inhibition of mitochondria bioenergetics in acetaminophen-induced liver injury.Mitochondrial function in ageing: coordination with signalling and transcriptional pathways.Energy metabolism and inflammation in brain aging and Alzheimer's disease.
P2860
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P2860
c-Jun N-terminal kinase regulates mitochondrial bioenergetics by modulating pyruvate dehydrogenase activity in primary cortical neurons.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@en
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@nl
type
label
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@en
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@nl
prefLabel
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@en
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@nl
P2093
P2860
P1476
c-Jun N-terminal kinase regula ...... y in primary cortical neurons.
@en
P2093
Derick Han
Enrique Cadenas
Philip Y Lam
Qiongqiong Zhou
P2860
P304
P356
10.1111/J.1471-4159.2007.04957.X
P407
P577
2007-10-18T00:00:00Z