Oxidative stress in the sympathetic premotor neurons contributes to sympathetic activation in renovascular hypertension.
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Effects of tempol and redox-cycling nitroxides in models of oxidative stress.Reactive Oxygen Species in the Paraventricular Nucleus of the Hypothalamus Alter Sympathetic Activity During Metabolic SyndromeReactive Oxygen Species: Physiological and Physiopathological Effects on Synaptic PlasticitySuperoxide anions in paraventricular nucleus modulate adipose afferent reflex and sympathetic activity in ratsTime-dependent effects of training on cardiovascular control in spontaneously hypertensive rats: role for brain oxidative stress and inflammation and baroreflex sensitivity.NADPH oxidases in vascular pathology.Essential hypertension and oxidative stress: New insightsNatural antioxidants and hypertension: promise and challenges.Mesenchymal stem cells (MSC) prevented the progression of renovascular hypertension, improved renal function and architecture.Chronic estradiol-17β exposure increases superoxide production in the rostral ventrolateral medulla and causes hypertension: reversal by resveratrol.Importance of rostral ventrolateral medulla neurons in determining efferent sympathetic nerve activity and blood pressurePVN adenovirus-siRNA injections silencing either NOX2 or NOX4 attenuate aldosterone/NaCl-induced hypertension in mice.Exercise training normalizes an increased neuronal excitability of NTS-projecting neurons of the hypothalamic paraventricular nucleus in hypertensive rats.Cardiac-autonomic imbalance and baroreflex dysfunction in the renovascular Angiotensin-dependent hypertensive mouse.Macrophage migration inhibitory factor in the nucleus of solitary tract decreases blood pressure in SHRs.DNA damage and augmented oxidative stress in bone marrow mononuclear cells from Angiotensin-dependent hypertensive mice.Membrane trafficking of NADPH oxidase p47(phox) in paraventricular hypothalamic neurons parallels local free radical production in angiotensin II slow-pressor hypertensionRenovascular hypertension leads to DNA damage and apoptosis in bone marrow cells.Neural Control of Non-vasomotor Organs in Hypertension.Predictors of mean arterial pressure morning rate of rise and power function in subjects undergoing ambulatory blood pressure recording.The role of oxidative stress in renovascular hypertension.Oxidative stress in the cardiovascular center has a pivotal role in the sympathetic activation in hypertension.Angiotensin-II-induced reactive oxygen species along the SFO-PVN-RVLM pathway: implications in neurogenic hypertension.Regulation of the sympathetic nervous system by nitric oxide and oxidative stress in the rostral ventrolateral medulla: 2012 Academic Conference Award from the Japanese Society of Hypertension.Effects of mesenchymal stem cells in renovascular hypertension.Mechanisms of renal sympathetic activation in renovascular hypertension.Mesenchymal stem cells and chronic renal artery stenosis.Compromised blood-brain barrier permeability: novel mechanism by which circulating angiotensin II signals to sympathoexcitatory centres during hypertension.Factors Responsible for Obesity-Related Hypertension.Maternal protein restriction induced-hypertension is associated to oxidative disruption at transcriptional and functional levels in the medulla oblongata.Direct vascular actions of quercetin in aorta from renal hypertensive rats.Increased expression of telomere-regulating genes in endurance athletes with long leukocyte telomeres.Exercise training restores oxidative stress and nitric oxide synthases in the rostral ventrolateral medulla of renovascular hypertensive rats.Sympathetic Overactivity in Chronic Kidney Disease: Consequences and Mechanisms.Sympathetic renal innervation and resistant hypertension.Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Activated intrarenal renin-angiotensin system is correIn vivo bioluminescence imaging reveals redox-regulated activator protein-1 activation in paraventricular nucleus of mice with renovascular hypertension.Oxidative stress in the brain causes hypertension via sympathoexcitation.Role of the NADPH oxidases in the subfornical organ in angiotensin II-induced hypertension.Increased expression of angiotensin II type 2 receptors in the solitary-vagal complex blunts renovascular hypertension.
P2860
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P2860
Oxidative stress in the sympathetic premotor neurons contributes to sympathetic activation in renovascular hypertension.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Oxidative stress in the sympat ...... in renovascular hypertension.
@en
Oxidative stress in the sympat ...... in renovascular hypertension.
@nl
type
label
Oxidative stress in the sympat ...... in renovascular hypertension.
@en
Oxidative stress in the sympat ...... in renovascular hypertension.
@nl
prefLabel
Oxidative stress in the sympat ...... in renovascular hypertension.
@en
Oxidative stress in the sympat ...... in renovascular hypertension.
@nl
P2093
P2860
P50
P356
P1476
Oxidative stress in the sympat ...... n in renovascular hypertension
@en
P2093
Bruno A Carillo
Elizabeth B Oliveira-Sales
Miriam S Dolnikoff
Mirian A Boim
P2860
P304
P356
10.1038/AJH.2009.17
P577
2009-02-19T00:00:00Z