α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

@en

α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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P1476

α1A-Subtype adrenergic agonist therapy for the failing right ventricle.

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P2093

Anthony J Baker

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David H Lovett

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Guanying Wang

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Patrick M Cowley

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Paul C Simpson

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Philip M Swigart

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P2860

Molecular Mechanisms of Right Ventricular Failure

Contrasting inotropic responses to alpha1-adrenergic receptor stimulation in left versus right ventricular myocardium

A novel intracellular isoform of matrix metalloproteinase-2 induced by oxidative stress activates innate immunity

Activation and modulation of 72kDa matrix metalloproteinase-2 by peroxynitrite and glutathione.

Heart failure switches the RV alpha1-adrenergic inotropic response from negative to positive.

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart.

A N-terminal truncated intracellular isoform of matrix metalloproteinase-2 impairs contractility of mouse myocardium.

Myofilament dysfunction contributes to impaired myocardial contraction in the infarct border zone

N-terminal truncated intracellular matrix metalloproteinase-2 induces cardiomyocyte hypertrophy, inflammation and systolic heart failure.

Chronic pulmonary artery pressure elevation is insufficient to explain right heart failure.

P304

H1109-H1118

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P31

scholarly article

P356

10.1152/AJPHEART.00153.2017

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P478

313

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P50

Sunil K Joshi

P577

2017-08-19T00:00:00Z

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P698

28822963

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P932

5814653

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