about
vProtein: identifying optimal amino acid complements from plant-based foodsMilk--A Nutrient System of Mammalian Evolution Promoting mTORC1-Dependent TranslationThe pathogenic role of persistent milk signaling in mTORC1- and milk-microRNA-driven type 2 diabetes mellitusOxidative stress and autophagy: the clash between damage and metabolic needsE2F1 regulates cellular growth by mTORC1 signaling.The general amino acid control pathway regulates mTOR and autophagy during serum/glutamine starvationPersistent hyperinsulinemic hypoglycemia of infancy: constitutive activation of the mTOR pathway with associated exocrine-islet transdifferentiation and therapeutic implicationsProton-assisted amino acid transporter PAT1 complexes with Rag GTPases and activates TORC1 on late endosomal and lysosomal membranes.Milk consumption during pregnancy increases birth weight, a risk factor for the development of diseases of civilizationThe mTORC1/4E-BP pathway coordinates hemoglobin production with L-leucine availability.Target of rapamycin (TOR) in nutrient signaling and growth controlDrosophila expresses a CD98 transporter with an evolutionarily conserved structure and amino acid-transport properties.Inhibition of glucose metabolism prevents glycosylation of the glutamine transporter ASCT2 and promotes compensatory LAT1 upregulation in leukemia cells.Blockade of Glutamine Synthetase Enhances Inflammatory Response in Microglial Cells.Mammalian target of rapamycin and the kidney. I. The signaling pathway.mTOR, metabolism, and the regulation of T-cell differentiation and function.L-Leucine induces growth arrest and persistent ERK activation in glioma cells.Inhibition of mTOR by temsirolimus contributes to prolonged survival of mice with pleural dissemination of non-small-cell lung cancer cells.Proton-assisted amino-acid transporters are conserved regulators of proliferation and amino-acid-dependent mTORC1 activation.The role of LAT1 in (18)F-DOPA uptake in malignant gliomas.Suppression of mTOR via Akt-dependent and -independent mechanisms in selenium-treated colon cancer cells: involvement of AMPKalpha1.
P2860
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P2860
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
An amino acid shuffle activates mTORC1.
@en
An amino acid shuffle activates mTORC1.
@nl
type
label
An amino acid shuffle activates mTORC1.
@en
An amino acid shuffle activates mTORC1.
@nl
prefLabel
An amino acid shuffle activates mTORC1.
@en
An amino acid shuffle activates mTORC1.
@nl
P1433
P1476
An amino acid shuffle activates mTORC1.
@en
P2093
Adiel Cohen
Michael N Hall
P304
P356
10.1016/J.CELL.2009.01.021
P407
P577
2009-02-01T00:00:00Z