Free cholesterol accumulation in hepatic stellate cells: mechanism of liver fibrosis aggravation in nonalcoholic steatohepatitis in mice. - Wikidata - awgldk - TriplyDB

Free cholesterol accumulation in hepatic stellate cells: mechanism of liver fibrosis aggravation in nonalcoholic steatohepatitis in mice.

Free cholesterol accumulation in hepatic stellate cells: mechanism of liver fibrosis aggravation in nonalcoholic steatohepatitis in mice.

http://www.wikidata.org/entity/Q46184549

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Evolving Insights on Metabolism, Autophagy, and Epigenetics in Liver Myofibroblasts Hepatic Stellate Cells and microRNAs in Pathogenesis of Liver Fibrosis Composite prognostic models across the non-alcoholic fatty liver disease spectrum: Clinical application in developing countries Non-alcoholic fatty liver disease: what has changed in the treatment since the beginning?Multiple hits, including oxidative stress, as pathogenesis and treatment target in non-alcoholic steatohepatitis (NASH)Molecular Pathogenesis of NASH Molecular mechanisms and new treatment strategies for non-alcoholic steatohepatitis (NASH)Dicer1/miR-29/HMGCR axis contributes to hepatic free cholesterol accumulation in mouse non-alcoholic steatohepatitis.The Role of Lipid and Lipoprotein Metabolism in Non-Alcoholic Fatty Liver Disease.Pathobiology of liver fibrosis: a translational success story Elevated free cholesterol in a p62 overexpression model of non-alcoholic steatohepatitis.TM6SF2 rs58542926 influences hepatic fibrosis progression in patients with non-alcoholic fatty liver disease.Recent Advances in Understanding of NASH: MicroRNAs as Both Biochemical Markers and Players.A Combination of Mitochondrial Oxidative Stress and Excess Fat/Calorie Intake Accelerates Steatohepatitis by Enhancing Hepatic CC Chemokine Production in Mice.Efficacy of probiotics in non-alcoholic fatty liver disease in adult and children: A meta-analysis of randomized controlled trials.Ethanol and High Cholesterol Diet Causes Severe Steatohepatitis and Early Liver Fibrosis in Mice.Ezetimibe for the treatment of non-alcoholic fatty liver disease: A meta-analysis.Saturated fat and cholesterol are critical to inducing murine metabolic syndrome with robust nonalcoholic steatohepatitis.Niemann-Pick Type C2 Protein Mediates Hepatic Stellate Cells Activation by Regulating Free Cholesterol Accumulation Liver Cholesterol Overload Aggravates Obstructive Cholestasis by Inducing Oxidative Stress and Premature Death in Mice Cytochrome P450-2E1 promotes fast food-mediated hepatic fibrosis.FOXO transcription factors protect against the diet-induced fatty liver disease.Revisiting the metabolic syndrome and paving the way for microRNAs in non-alcoholic fatty liver disease.miRNA in hepatocellular carcinoma.Therapeutic application of GPR119 ligands in metabolic disorders.Review article: emerging anti-fibrotic therapies in the treatment of non-alcoholic steatohepatitis.The role of iNOS in cholesterol-induced liver fibrosis.The independent predictors of non-alcoholic steatohepatitis and its individual histological features.: Insulin resistance, serum uric acid, metabolic syndrome, alanine aminotransferase and serum total cholesterol are a clue to pathogenesis and candi Hepatic stellate cells as key target in liver fibrosis.Pigment Epithelium-Derived Factor (PEDF) Prevents Hepatic Fat Storage, Inflammation, and Fibrosis in Dietary Steatohepatitis of Mice.Transient Hepatic Overexpression of Insulin-Like Growth Factor 2 Induces Free Cholesterol and Lipid Droplet Formation.Cholesterol 7α-hydroxylase protects the liver from inflammation and fibrosis by maintaining cholesterol homeostasis.A novel diet-induced murine model of steatohepatitis with fibrosis for screening and evaluation of drug candidates for nonalcoholic steatohepatitis.Intracellular hepatitis B virus increases hepatic cholesterol deposition in alcoholic fatty liver via hepatitis B core protein.Cholesterol is a significant risk factor for non-alcoholic steatohepatitis.BMP Signalling at the Crossroad of Liver Fibrosis and Regeneration.Vitamin A-coupled liposome system targeting free cholesterol accumulation in hepatic stellate cells offers a beneficial therapeutic strategy for liver fibrosis.Lipopolysaccharide mediates hepatic stellate cell activation by regulating autophagy and retinoic acid signaling.SREBP-regulated lipid metabolism: convergent physiology - divergent pathophysiology.Activation of Insulin-PI3K/Akt-p70S6K Pathway in Hepatic Stellate Cells Contributes to Fibrosis in Nonalcoholic Steatohepatitis.

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Free cholesterol accumulation in hepatic stellate cells: mechanism of liver fibrosis aggravation in nonalcoholic steatohepatitis in mice.

http://www.wikidata.org/entity/Q46184549

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2013 nî lūn-bûn

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2013年學術文章

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Free cholesterol accumulation ...... holic steatohepatitis in mice.

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Free cholesterol accumulation ...... holic steatohepatitis in mice.

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Free cholesterol accumulation ...... holic steatohepatitis in mice.

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Free cholesterol accumulation ...... holic steatohepatitis in mice.

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Free cholesterol accumulation ...... holic steatohepatitis in mice.

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Atsushi Kawaguchi

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Chie Kurihara

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Chikako Watanabe

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Hidetsugu Saito

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Hirokazu Sato

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Hirokazu Yokoyama

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Hirotoshi Ebinuma

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Katsuyoshi Shimamura

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Kazuo Sugiyama

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Kazuyuki Narimatsu

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P2860

A proteolytic pathway that controls the cholesterol content of membranes, cells, and blood

Combined analysis of oligonucleotide microarray data from transgenic and knockout mice identifies direct SREBP target genes

Triad3A, an E3 ubiquitin-protein ligase regulating Toll-like receptors

TLR4 enhances TGF-beta signaling and hepatic fibrosis

CYP2E1 and CYP4A as microsomal catalysts of lipid peroxides in murine nonalcoholic steatohepatitis

27-Hydroxycholesterol: the first identified endogenous SERM

Tumour necrosis factor alpha signalling through activation of Kupffer cells plays an essential role in liver fibrosis of non-alcoholic steatohepatitis in mice.

Sterols regulate processing of carbohydrate chains of wild-type SREBP cleavage-activating protein (SCAP), but not sterol-resistant mutants Y298C or D443N.

SREBP in signal transduction: cholesterol metabolism and beyond.

Cyclodextrin overcomes deficient lysosome-to-endoplasmic reticulum transport of cholesterol in Niemann-Pick type C cells.

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10.1002/HEP.26604

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23832448

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Steatohepatitis