In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role? - Wikidata - awgldk - TriplyDB

In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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2015年学术文章

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In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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In reply: Reoperation for recurrent high-grade glioma: does tumor genetics play a role?

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Mitchel S Berger

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Shawn L Hervey-Jumper

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O(6)-methylguanine DNA-methyltransferase methylation status can change between first surgery for newly diagnosed glioblastoma and second surgery for recurrence: clinical implications.

Methylation profiling identifies 2 groups of gliomas according to their tumorigenesis

Cancer genomic research at the crossroads: realizing the changing genetic landscape as intratumoral spatial and temporal heterogeneity becomes a confounding factor.

Concurrent hypermethylation of DNMT1, MGMT and EGFR genes in progression of gliomas.

Reoperation for recurrent high-grade glioma: a current perspective of the literature.

Molecular heterogeneity of glioblastoma and its clinical relevance.

Molecular and cellular heterogeneity: the hallmark of glioblastoma.

Prognostic value of 1p, 19q, 9p, 10q, and EGFR-FISH analyses in recurrent oligodendrogliomas.

Promoter methylation and expression of MGMT and the DNA mismatch repair genes MLH1, MSH2, MSH6 and PMS2 in paired primary and recurrent glioblastomas.

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