Dissociation of E-cadherin and beta-catenin in a mouse model of total parenteral nutrition: a mechanism for the loss of epithelial cell proliferation and villus atrophy.
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hPSC-derived lung and intestinal organoids as models of human fetal tissueIntestinal microbial diversity and perioperative complicationsProceedings of the 2013 A.S.P.E.N. Research workshop: the interface between nutrition and the gut microbiome: implications and applications for human health [corrected].Intestinal epithelial cell apoptosis and loss of barrier function in the setting of altered microbiota with enteral nutrient deprivation.Enteral nutrient deprivation in patients leads to a loss of intestinal epithelial barrier functionDistraction-induced intestinal enterogenesis: preservation of intestinal function and lengthening after reimplantation into normal jejunumTPN-associated intestinal epithelial cell atrophy is modulated by TLR4/EGF signaling pathways.Epidermal growth factor/TNF-α transactivation modulates epithelial cell proliferation and apoptosis in a mouse model of parenteral nutrition.Loss of MYO5B in mice recapitulates Microvillus Inclusion Disease and reveals an apical trafficking pathway distinct to neonatal duodenum.Total parenteral nutrition-associated lamina propria inflammation in mice is mediated by a MyD88-dependent mechanism.Diet-dependent, microbiota-independent regulation of IL-10-producing lamina propria macrophages in the small intestine.Decreased phospho-Akt signaling in a mouse model of total parenteral nutrition: a potential mechanism for the development of intestinal mucosal atrophyDistraction-induced intestinal growth: the role of mechanotransduction mechanisms in a mouse model of short bowel syndrome.Glutamate prevents intestinal atrophy via luminal nutrient sensing in a mouse model of total parenteral nutrition.Bacterial nutrient foraging in a mouse model of enteral nutrient deprivation: insight into the gut origin of sepsis.Tight Junction Ultrastructure Alterations in a Mouse Model of Enteral Nutrient Deprivation.The bone morphogenetic protein signaling pathway is upregulated in a mouse model of total parenteral nutritionLoss of enteral nutrition in a mouse model results in intestinal epithelial barrier dysfunction.Angiotensin converting enzyme-inhibitor reduces colitis severity in an IL-10 knockout model.Parenteral Nutrition-Associated Liver Disease: The Role of the Gut MicrobiotaTrafficking Ion Transporters to the Apical Membrane of Polarized Intestinal Enterocytes.Olive Oil-Supplemented Lipid Emulsion Induces CELF1 Expression and Promotes Apoptosis in Caco-2 Cells.Effects on varying intravenous lipid emulsions on the small bowel epithelium in a mouse model of parenteral nutrition.Tumour necrosis factor--induced loss of intestinal barrier function requires TNFR1 and TNFR2 signalling in a mouse model of total parenteral nutrition.Role of glutamine in the mediation of E-cadherin, p120-catenin and inflammation in ventilator-induced lung injury.
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Dissociation of E-cadherin and beta-catenin in a mouse model of total parenteral nutrition: a mechanism for the loss of epithelial cell proliferation and villus atrophy.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@en
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@nl
type
label
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@en
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@nl
prefLabel
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@en
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@nl
P2093
P2860
P1476
Dissociation of E-cadherin and ...... liferation and villus atrophy.
@en
P2093
Daniel H Teitelbaum
Xiaoyi Sun
Yongjia Feng
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P304
P356
10.1113/JPHYSIOL.2008.162719
P407
P577
2008-12-08T00:00:00Z