Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
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Cutis laxa: intersection of elastic fiber biogenesis, TGFβ signaling, the secretory pathway and metabolismSLC2A10 genetic polymorphism predicts development of peripheral arterial disease in patients with type 2 diabetes. SLC2A10 and PAD in type 2 diabetes.Glioma morphology and tumor-induced vascular alterations revealed in seven rodent glioma models by in vivo magnetic resonance imaging and angiography.Mitochondrial GLUT10 facilitates dehydroascorbic acid import and protects cells against oxidative stress: mechanistic insight into arterial tortuosity syndrome.The pathobiology of vascular malformations: insights from human and model organism genetics.GLUT10 is required for the development of the cardiovascular system and the notochord and connects mitochondrial function to TGFβ signalingTwisted blood vessels: symptoms, etiology and biomechanical mechanismsEffect of Hyperglycemia on Gene Expression during Early Organogenesis in Mice.GLUT10 deficiency leads to oxidative stress and non-canonical αvβ3 integrin-mediated TGFβ signalling associated with extracellular matrix disarray in arterial tortuosity syndrome skin fibroblastsLessons on the pathogenesis of aneurysm from heritable conditionsRandom mutagenesis of the mouse genome: a strategy for discovering gene function and the molecular basis of disease.Glucose transporter 10 and arterial tortuosity syndrome: the vitamin C connection.Hepatic expression and cellular distribution of the glucose transporter family.Proteomics in aortic aneurysm--what have we learnt so far?Review of molecular and mechanical interactions in the aortic valve and aorta: implications for the shared pathogenesis of aortic valve disease and aortopathy.Function of Ltbp-4L and fibulin-4 in survival and elastogenesis in mice.Genes Associated with Thoracic Aortic Aneurysm and Dissection: An Update and Clinical Implications.Glucose transporter type 10-lacking in arterial tortuosity syndrome-facilitates dehydroascorbic acid transport.Autosomal and X chromosome structural variants are associated with congenital heart defects in Turner syndrome: The NHLBI GenTAC registry.
P2860
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P2860
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@en
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@nl
type
label
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@en
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@nl
prefLabel
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@en
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@nl
P2093
P2860
P356
P1476
Mutations in the SLC2A10 gene cause arterial abnormalities in mice.
@en
P2093
Chao-Hung Cheng
Chen Chang
Huei-Ju Pan
Nagham George Abd-Al-Ahad Sabbagha
Tateki Kikuchi
Yen-Hui Chen
Yi-Ching Lee
P2860
P304
P356
10.1093/CVR/CVN319
P577
2008-11-21T00:00:00Z