Reactive oxygen species damage drives cardiac and mitochondrial dysfunction following acute nano-titanium dioxide inhalation exposure.

about

Reactive oxygen species damage drives cardiac and mitochondrial dysfunction following acute nano-titanium dioxide inhalation exposure.

Item

http://www.wikidata.org/entity/Q46240480

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@en

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@nl

type

Item

label

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@en

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@nl

prefLabel

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@en

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@nl

P1476

Reactive oxygen species damage ...... m dioxide inhalation exposure.

@en

P2093

Alaeddin Abukabda

http://www.w3.org/2001/XMLSchema#string

Andrya J Durr

http://www.w3.org/2001/XMLSchema#string

Cody E Nichols

http://www.w3.org/2001/XMLSchema#string

Danielle L Shepherd

http://www.w3.org/2001/XMLSchema#string

Jinghai Yi

http://www.w3.org/2001/XMLSchema#string

John M Hollander

http://www.w3.org/2001/XMLSchema#string

Quincy A Hathaway

http://www.w3.org/2001/XMLSchema#string

Timothy R Nurkiewicz

http://www.w3.org/2001/XMLSchema#string

P2860

Nanoparticle inhalation impairs coronary microvascular reactivity via a local reactive oxygen species-dependent mechanism

Nanoparticle inhalation impairs endothelium-dependent vasodilation in subepicardial arterioles

Cardiac and mitochondrial dysfunction following acute pulmonary exposure to mountaintop removal mining particulate matter

Acute pulmonary dose-responses to inhaled multi-walled carbon nanotubes

Pulmonary exposure of rats to ultrafine titanium dioxide enhances cardiac protein phosphorylation and substance P synthesis in nodose ganglia

The role of nodose ganglia in the regulation of cardiovascular function following pulmonary exposure to ultrafine titanium dioxide

Whole-body nanoparticle aerosol inhalation exposure

Pulmonary nanoparticle exposure disrupts systemic microvascular nitric oxide signaling

Quinones and aromatic chemical compounds in particulate matter induce mitochondrial dysfunction: implications for ultrafine particle toxicity

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

P304

32-48

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1080/17435390.2017.1416202

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Dharendra Thapa

P577

2017-12-15T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29243970

http://www.w3.org/2001/XMLSchema#string

P921

Béésh doo Ńdiniichxíihii

P932

5777890

http://www.w3.org/2001/XMLSchema#string