N-methyl-d-aspartic acid receptor antagonist-induced frequency oscillations in mice recreate pattern of electrophysiological deficits in schizophrenia.
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GABAB-mediated rescue of altered excitatory-inhibitory balance, gamma synchrony and behavioral deficits following constitutive NMDAR-hypofunction.Impact of ketamine on neuronal network dynamics: translational modeling of schizophrenia-relevant deficits.Neuromagnetic oscillations predict evoked-response latency delays and core language deficits in autism spectrum disorders.Phencyclidine Disrupts the Auditory Steady State Response in RatsSensory processing in autism spectrum disorders and Fragile X syndrome-From the clinic to animal models.The auditory steady-state response (ASSR): a translational biomarker for schizophrenia.Reversal of evoked gamma oscillation deficits is predictive of antipsychotic activity with a unique profile for clozapine.Pyramidal cell selective ablation of N-methyl-D-aspartate receptor 1 causes increase in cellular and network excitability.Juvenile antioxidant treatment prevents adult deficits in a developmental model of schizophreniaJuvenile exposure to ketamine causes delayed emergence of EEG abnormalities during adulthood in miceBrain state-dependent abnormal LFP activity in the auditory cortex of a schizophrenia mouse model.Knockout of NMDA receptors in parvalbumin interneurons recreates autism-like phenotypes.Electrophysiological and behavioral responses to ketamine in mice with reduced Akt1 expression.How Nox2-containing NADPH oxidase affects cortical circuits in the NMDA receptor antagonist model of schizophrenia.Mice with reduced NMDA receptor expression: more consistent with autism than schizophrenia?Prolonged Ketamine Effects in Sp4 Hypomorphic Mice: Mimicking Phenotypes of Schizophrenia.Augmented gamma band auditory steady-state responses: support for NMDA hypofunction in schizophreniaAcute administration of typical and atypical antipsychotics reduces EEG γ power, but only the preclinical compound LY379268 reduces the ketamine-induced rise in γ power.Abnormalities of neuronal oscillations and temporal integration to low- and high-frequency auditory stimulation in schizophreniaA critical role for NMDA receptors in parvalbumin interneurons for gamma rhythm induction and behavior.Steady state responses: electrophysiological assessment of sensory function in schizophrenia.Parvalbumin cell ablation of NMDA-R1 causes increased resting network excitability with associated social and self-care deficits.Alterations in High-Frequency Neuronal Oscillations in a Cynomolgus Macaque Test of Sustained Attention Following NMDA Receptor Antagonism.Delayed emergence of behavioral and electrophysiological effects following juvenile ketamine exposure in miceA Neurophysiological Perspective on a Preventive Treatment against Schizophrenia Using Transcranial Electric Stimulation of the Corticothalamic PathwayAmplitude of low-frequency oscillations in schizophrenia: a resting state fMRI study.Cannabinoids and Vanilloids in Schizophrenia: Neurophysiological Evidence and Directions for Basic Research.Reviewing the ketamine model for schizophrenia.Ketamine and phencyclidine: the good, the bad and the unexpected.Gamma and delta neural oscillations and association with clinical symptoms under subanesthetic ketamineCognition, dopamine and bioactive lipids in schizophrenia.A canonical circuit for generating phase-amplitude coupling.Baseline gamma power during auditory steady-state stimulation in schizophreniaMeCP2+/- mouse model of RTT reproduces auditory phenotypes associated with Rett syndrome and replicate select EEG endophenotypes of autism spectrum disorder.Ketamine Dysregulates the Amplitude and Connectivity of High-Frequency Oscillations in Cortical-Subcortical Networks in Humans: Evidence From Resting-State Magnetoencephalography-RecordingsRole of N-Methyl-D-Aspartate Receptors in Action-Based Predictive Coding Deficits in Schizophrenia.First and second generation antipsychotics influence hippocampal gamma oscillations by interactions with 5-HT3 and D3 receptorsMemantine Improves Attentional Processes in Fragile X-Associated Tremor/Ataxia Syndrome: Electrophysiological Evidence from a Randomized Controlled Trial.Relationship of resting brain hyperconnectivity and schizophrenia-like symptoms produced by the NMDA receptor antagonist ketamine in humans.Dopamine, cognitive function, and gamma oscillations: role of D4 receptors.
P2860
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P2860
N-methyl-d-aspartic acid receptor antagonist-induced frequency oscillations in mice recreate pattern of electrophysiological deficits in schizophrenia.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@en
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@nl
type
label
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@en
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@nl
prefLabel
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@en
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@nl
P2093
P1433
P1476
N-methyl-d-aspartic acid recep ...... cal deficits in schizophrenia.
@en
P2093
B I Turetsky
C R Maxwell
D Contreras
L H Finkel
M T Lazarewicz
R S Ehrlichman
P304
P356
10.1016/J.NEUROSCIENCE.2008.10.031
P407
P577
2008-10-28T00:00:00Z