Monocytes enhance neutrophil-induced blister formation in an ex vivo model of bullous pemphigoid.

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The Immunometabolomic Interface Receptor Hydroxycarboxylic Acid Receptor 2 Mediates the Therapeutic Effects of Dimethyl Fumarate in Autoantibody-Induced Skin Inflammation

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Monocytes enhance neutrophil-induced blister formation in an ex vivo model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... o model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... o model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... o model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... o model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... o model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... o model of bullous pemphigoid.

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Monocytes enhance neutrophil-i ...... vo model of bullous pemphigoid

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E de Graauw

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L Borradori

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M P Horn

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P2860

Neutrophil elastase cleaves the murine hemidesmosomal protein BP180/type XVII collagen and generates degradation products that modulate experimental bullous pemphigoid

Reactive oxygen and nitrogen intermediates in the relationship between mammalian hosts and microbial pathogens

Fiji: an open-source platform for biological-image analysis.

The IL-8 release from cultured human keratinocytes, mediated by antibodies to bullous pemphigoid autoantigen 180, is inhibited by dapsone

Intracellular localization of the BCL-2 family member BOK and functional implications

A passive transfer model of the organ-specific autoimmune disease, bullous pemphigoid, using antibodies generated against the hemidesmosomal antigen, BP180.

Definitions and outcome measures for bullous pemphigoid: recommendations by an international panel of experts.

92-kD gelatinase is produced by eosinophils at the site of blister formation in bullous pemphigoid and cleaves the extracellular domain of recombinant 180-kD bullous pemphigoid autoantigen.

Exosomes as divine messengers: are they the Hermes of modern molecular oncology?

Nitric oxide and redox mechanisms in the immune response.

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1119-1130

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scholarly article

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10.1111/ALL.13376

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Hans-Uwe Simon

Shida Yousefi

Dagmar Simon

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2018-01-17T00:00:00Z

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29222810

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Monocytes enhance neutrophil-induced blister formation in an ex vivo model of bullous pemphigoid.

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Monocytes enhance neutrophil-induced blister formation in an ex vivo model of bullous pemphigoid.

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