Energy depletion and not ROS formation is a crucial step of glucolipotoxicity (GLTx) in pancreatic beta cells.

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Energy depletion and not ROS formation is a crucial step of glucolipotoxicity (GLTx) in pancreatic beta cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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Energy depletion and not ROS f ...... LTx) in pancreatic beta cells.

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Energy depletion and not ROS formation is a crucial step of glucolipotoxicity

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Energy depletion and not ROS f ...... LTx) in pancreatic beta cells.

@en

Energy depletion and not ROS formation is a crucial step of glucolipotoxicity

@nl

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Energy depletion and not ROS f ...... LTx) in pancreatic beta cells.

@en

Energy depletion and not ROS formation is a crucial step of glucolipotoxicity

@nl

P1476

Energy depletion and not ROS f ...... GLTx) in pancreatic beta cells

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P2093

Morgana Barroso Oquendo

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Nikolas Layer

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Peter Krippeit-Drews

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Rebecca Wagner

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P2860

Oxidative and endoplasmic reticulum stress in β-cell dysfunction in diabetes

Impaired gene and protein expression of exocytotic soluble N-ethylmaleimide attachment protein receptor complex proteins in pancreatic islets of type 2 diabetic patients.

JNK1 protects against glucolipotoxicity-mediated beta-cell apoptosis

Fatty acids and glucose in high concentration down-regulates ATP synthase beta-subunit protein expression in INS-1 cells.

Diabetic beta-cells can achieve self-protection against oxidative stress through an adaptive up-regulation of their antioxidant defenses.

GLP1-derived nonapeptide GLP1(28-36)amide protects pancreatic β-cells from glucolipotoxicity

Chronic glucolipotoxic conditions in pancreatic islets impair insulin secretion due to dysregulated calcium dynamics, glucose responsiveness and mitochondrial activity.

Isoprenylcysteine carboxyl methyltransferase facilitates glucose-induced Rac1 activation, ROS generation and insulin secretion in INS 832/13 β-cells.

Diabetes associated cell stress and dysfunction: role of mitochondrial and non-mitochondrial ROS production and activity.

Glucolipotoxicity: fuel excess and beta-cell dysfunction.

P2888

s00424-017-2094-8

P304

537-547

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P31

scholarly article

P356

10.1007/S00424-017-2094-8

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P433

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P478

470

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P50

Gisela Drews

P577

2017-12-07T00:00:00Z

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P698

29218453

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