Thin myelin sheaths as the hallmark of remyelination persist over time and preserve axon function.

about

Thin myelin sheaths as the hallmark of remyelination persist over time and preserve axon function.

Item

http://www.wikidata.org/entity/Q46273126

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Thin myelin sheaths as the hal ...... me and preserve axon function.

@en

Thin myelin sheaths as the hal ...... me and preserve axon function.

@nl

type

Item

label

Thin myelin sheaths as the hal ...... me and preserve axon function.

@en

Thin myelin sheaths as the hal ...... me and preserve axon function.

@nl

prefLabel

Thin myelin sheaths as the hal ...... me and preserve axon function.

@en

Thin myelin sheaths as the hal ...... me and preserve axon function.

@nl

P1476

Thin myelin sheaths as the hal ...... me and preserve axon function.

@en

P2093

Aimee T Broman

http://www.w3.org/2001/XMLSchema#string

Ian D Duncan

http://www.w3.org/2001/XMLSchema#string

Moones Heidari

http://www.w3.org/2001/XMLSchema#string

Rachel L Marik

http://www.w3.org/2001/XMLSchema#string

P2860

Promoting remyelination in multiple sclerosis-recent advances

Random-effects models for longitudinal data

Disruption of Cnp1 uncouples oligodendroglial functions in axonal support and myelination

Remyelination protects axons from demyelination-associated axon degeneration

From fish to man: understanding endogenous remyelination in central nervous system demyelinating diseases.

A mutation in the canine gene encoding folliculin-interacting protein 2 (FNIP2) associated with a unique disruption in spinal cord myelination.

Understanding CNS remyelination: clues from developmental and regeneration biology.

Oligodendroglia metabolically support axons and contribute to neurodegeneration.

Axonal damage: a key predictor of outcome in human CNS diseases.

Remyelination can be extensive in multiple sclerosis despite a long disease course.

P304

E9685-E9691

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.1714183114

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

114

http://www.w3.org/2001/XMLSchema#string

P577

2017-10-24T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29078396

http://www.w3.org/2001/XMLSchema#string

P932

5692595

http://www.w3.org/2001/XMLSchema#string