Inhibition of Kupffer cell-mediated early proinflammatory response with carbon monoxide in transplant-induced hepatic ischemia/reperfusion injury in rats. - Wikidata - awgldk - TriplyDB

Inhibition of Kupffer cell-mediated early proinflammatory response with carbon monoxide in transplant-induced hepatic ischemia/reperfusion injury in rats.

Inhibition of Kupffer cell-mediated early proinflammatory response with carbon monoxide in transplant-induced hepatic ischemia/reperfusion injury in rats.

http://www.wikidata.org/entity/Q46274424

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Role of NK, NKT cells and macrophages in liver transplantation Novel Action of Carotenoids on Non-Alcoholic Fatty Liver Disease: Macrophage Polarization and Liver Homeostasis Use of carbon monoxide in minimizing ischemia/reperfusion injury in transplantation Hydroxysafflor yellow A attenuates ischemia/reperfusion-induced liver injury by suppressing macrophage activation.The Carbon monoxide releasing molecule ALF-186 mediates anti-inflammatory and neuroprotective effects via the soluble guanylate cyclase ß1 in rats' retinal ganglion cells after ischemia and reperfusion injury Liver graft exposure to carbon monoxide during cold storage protects sinusoidal endothelial cells and ameliorates reperfusion injury in rats.Carbon monoxide induces hypothermia tolerance in Kupffer cells and attenuates liver ischemia/reperfusion injury in rats.Effects and mechanisms of store-operated calcium channel blockade on hepatic ischemia-reperfusion injury in rats.Down-Regulated Receptor Interacting Protein 140 Is Involved in Lipopolysaccharide-Preconditioning-Induced Inactivation of Kupffer Cells and Attenuation of Hepatic Ischemia Reperfusion Injury Functional role of monocytes and macrophages for the inflammatory response in acute liver injury.Hepatic macrophages in homeostasis and liver diseases: from pathogenesis to novel therapeutic strategies.Low-dose carbon monoxide inhibits progressive chronic allograft nephropathy and restores renal allograft function.Cell biology of ischemia/reperfusion injury.Carbon monoxide: endogenous mediator, potential diagnostic and therapeutic target.Carbon monoxide: an unusual drug.Macrophage plasticity and polarization in liver homeostasis and pathology.Ischemia/Reperfusion.Heme oxygenase-1 protects donor livers from ischemia/reperfusion injury: the role of Kupffer cells.Carbon monoxide ameliorates hepatic ischemia/reperfusion injury via sirtuin 1-mediated deacetylation of high-mobility group box 1 in rats.Carbon monoxide-releasing molecule-2 (CORM-2) attenuates acute hepatic ischemia reperfusion injury in rats.Heme oxygenase system in hepatic ischemia-reperfusion injury Emulsified isoflurane preconditioning protects isolated rat Kupffer cells against hypoxia/reoxygenation-induced injury.A novel mouse model of depletion of stellate cells clarifies their role in ischemia/reperfusion- and endotoxin-induced acute liver injury.Carbon monoxide enhances early liver regeneration in mice after hepatectomy.Protective effect of heme oxygenase-1 on hepatic ischemia-reperfusion injury through inhibition of platelet adhesion to the sinusoids.Myeloid Notch1 Deficiency Activates RhoA/ROCK Pathway and Aggravates Hepatocellular Damage In Mouse Ischemic Livers.Anti-inflammatory signaling during ex vivo liver perfusion improves the preservation of pig liver grafts before transplantation.Neutrophil extracellular traps in acrolein promoted hepatic ischemia reperfusion injury: Therapeutic potential of NOX2 and p38MAPK inhibitors.Subnormothermic ex vivo liver perfusion is a safe alternative to cold static storage for preserving standard criteria grafts.Contribution of alloantigens to hepatic ischemia/reperfusion injury: Roles of natural killer cells and innate immune recognition of nonself.Barrier-tissue macrophages: functional adaptation to environmental challenges.Serelaxin Induces Notch1 Signaling and Alleviates Hepatocellular Damage in Orthotopic Liver Transplantation.Iron-induced oxidative rat liver injury after non-heart-beating warm ischemia is mediated by tumor necrosis factor α and prevented by deferoxamine.Subnormothermic ex vivo liver perfusion reduces endothelial cell and bile duct injury after donation after cardiac death pig liver transplantation.Soluble fibrinogen-like protein 2 ameliorates acute rejection of liver transplantation in rat via inducing Kupffer cells M2 polarization.Dual Effect of Hepatic Macrophages on Liver Ischemia and Reperfusion Injury during Liver Transplantation.

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Inhibition of Kupffer cell-mediated early proinflammatory response with carbon monoxide in transplant-induced hepatic ischemia/reperfusion injury in rats.

http://www.wikidata.org/entity/Q46274424

description

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Amin Afrazi

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Atsunori Nakao

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Atsushi Ikeda

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Chandrashekhar Gandhi

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Daisuke Tokita

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David A Geller

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Donna B Stolz

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Koji Tomiyama

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Noriko Murase

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Shinya Ueki

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P2860

Kupffer cell-independent acute hepatocellular oxidative stress and decreased bile formation in post-cold-ischemic rat liver.

Kupffer cells alter organic anion transport through multidrug resistance protein 2 in the post-cold ischemic rat liver.

Carbon monoxide: a putative neural messenger.

'Green mice' as a source of ubiquitous green cells.

Kupffer cell-dependent TNF-alpha signaling mediates injury in the arterialized small-for-size liver transplantation in the mouse.

Hepatic ischemia reperfusion injury: pathogenic mechanisms and basis for hepatoprotection.

Carbon monoxide inhalation protects rat intestinal grafts from ischemia/reperfusion injury.

Sinusoidal endothelial cell repopulation following ischemia/reperfusion injury in rat liver transplantation.

Past and future approaches to ischemia-reperfusion lesion associated with liver transplantation.

CCAAT/enhancer-binding protein mediates carbon monoxide-induced suppression of cyclooxygenase-2.

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1608-1620

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10.1002/HEP.22482

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18972563

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reperfusion injury

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