Pendrin in the mouse kidney is primarily regulated by Cl- excretion but also by systemic metabolic acidosis.
about
Collecting duct intercalated cell function and regulationA mouse model for distal renal tubular acidosis reveals a previously unrecognized role of the V-ATPase a4 subunit in the proximal tubule.Bile acids trigger cholemic nephropathy in common bile-duct-ligated mice.Proliferation of acid-secretory cells in the kidney during adaptive remodelling of the collecting duct.Insights into acidosis-induced regulation of SLC26A4 (pendrin) and SLC4A9 (AE4) transporters using three-dimensional morphometric analysis of β-intercalated cells.Regulation of two renal chloride transporters, AE1 and pendrin, by electrolytes and aldosteroneDirect physical contact between intercalated cells in the distal convoluted tubule and the afferent arteriole in mouse kidneys.Colocalization of the (Pro)renin Receptor/Atp6ap2 with H+-ATPases in Mouse Kidney but Prorenin Does Not Acutely Regulate Intercalated Cell H+-ATPase Activity.Altered regulation of renal Acid base transporters in response to ammonium chloride loading in rats.A new look at electrolyte transport in the distal tubuleSDF1 induction by acidosis from principal cells regulates intercalated cell subtype distribution.Bicarbonate promotes BK-α/β4-mediated K excretion in the renal distal nephronProximal tubule Na+/H+ exchanger activity in adult NHE8-/-, NHE3-/-, and NHE3-/-/NHE8-/- mice.Haploinsufficiency of the ammonia transporter Rhcg predisposes to chronic acidosis: Rhcg is critical for apical and basolateral ammonia transport in the mouse collecting ductAngiotensin II stimulates H⁺-ATPase activity in intercalated cells from isolated mouse connecting tubules and cortical collecting ductsAcetazolamide Attenuates Lithium-Induced Nephrogenic Diabetes InsipidusThe anion exchanger pendrin (SLC26A4) and renal acid-base homeostasis.Uriniferous tubule: structural and functional organization.Acute regulated expression of pendrin in human urinary exosomes.Mineralocorticoid receptor as a therapeutic target in chronic kidney disease and hypertension.Role of Pendrin in Acid-base Balance.Pathophysiology, diagnosis and treatment of inherited distal renal tubular acidosis.DOCA sensitive pendrin expression in kidney, heart, lung and thyroid tissues.Impact of bicarbonate, ammonium chloride, and acetazolamide on hepatic and renal SLC26A4 expression.A fate-mapping approach reveals the composite origin of the connecting tubule and alerts on "single-cell"-specific KO model of the distal nephron.
P2860
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P2860
Pendrin in the mouse kidney is primarily regulated by Cl- excretion but also by systemic metabolic acidosis.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@en
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@nl
type
label
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@en
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@nl
prefLabel
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@en
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@nl
P2093
P2860
P1476
Pendrin in the mouse kidney is ...... y systemic metabolic acidosis.
@en
P2093
Carsten A Wagner
Giovambattista Capasso
Paola Capuano
Patricia Hafner
Rosa Grimaldi
P2860
P304
P356
10.1152/AJPCELL.00419.2008
P577
2008-10-29T00:00:00Z