N-acetylglucosamine: more than a silent partner in insulin resistance.

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N-acetylglucosamine: more than a silent partner in insulin resistance.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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N-acetylglucosamine: more than a silent partner in insulin resistance.

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N-acetylglucosamine: more than a silent partner in insulin resistance.

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N-acetylglucosamine: more than a silent partner in insulin resistance.

@en

N-acetylglucosamine: more than a silent partner in insulin resistance.

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N-acetylglucosamine: more than a silent partner in insulin resistance.

@en

N-acetylglucosamine: more than a silent partner in insulin resistance.

@nl

P1476

N-acetylglucosamine: more than a silent partner in insulin resistance.

@en

P2093

Geoffrey G Hesketh

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James W Dennis

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P2860

SLC38A9 is a component of the lysosomal amino acid sensing machinery that controls mTORC1

Metabolism. Lysosomal amino acid transporter SLC38A9 signals arginine sufficiency to mTORC1

mTOR: from growth signal integration to cancer, diabetes and ageing

A branched-chain amino acid-related metabolic signature that differentiates obese and lean humans and contributes to insulin resistance

Deep congenic analysis identifies many strong, context-dependent QTLs, one of which, Slc35b4, regulates obesity and glucose homeostasis

The genetic architecture of type 2 diabetes

Bidirectional transport of amino acids regulates mTOR and autophagy

Glycolysis and glutaminolysis cooperatively control T cell function by limiting metabolite supply to N-glycosylation.

Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis.

Baleen whales host a unique gut microbiome with similarities to both carnivores and herbivores

P304

595-598

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P31

scholarly article

P356

10.1093/GLYCOB/CWX035

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P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-07-01T00:00:00Z

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P698

29048482

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P932

5458536

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