Adenosine derived from ecto-nucleotidases in calcific aortic valve disease promotes mineralization through A2a adenosine receptor.
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MicroRNAs in Valvular Heart Diseases: Potential Role as Markers and Actors of Valvular and Cardiac RemodelingNucleotide Catabolism on the Surface of Aortic Valve Xenografts; Effects of Different Decellularization Strategies.Regulation of CD73 in the development of lower limb atherosclerosis.Polymorphism in exon 6 of the human NT5E gene is associated with aortic valve calcification.Editorial highlights from Cardiovascular Research.OxLDL-derived lysophosphatidic acid promotes the progression of aortic valve stenosis through a LPAR1-RhoA-NF-κB pathway.Oxidized low-density lipoproteins enhance expression and activity of CD39 and CD73 in the human aortic valve endothelium.
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P2860
Adenosine derived from ecto-nucleotidases in calcific aortic valve disease promotes mineralization through A2a adenosine receptor.
description
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name
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@en
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@nl
type
label
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@en
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@nl
prefLabel
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@en
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@nl
P2093
P2860
P356
P1476
Adenosine derived from ecto-nu ...... hrough A2a adenosine receptor.
@en
P2093
Ablajan Mahmut
Fayez Hadji
Marie-Chloé Boulanger
Patrick Mathieu
Rihab Bouchareb
P2860
P304
P356
10.1093/CVR/CVV027
P577
2015-02-02T00:00:00Z