Establishment of Singleton-Merten syndrome pulp cells: evidence of mineralization dysregulation.

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A specific IFIH1 gain-of-function mutation causes Singleton-Merten syndrome.Genetically defined autoinflammatory diseases.RIG-I-Like Receptor Signaling in Singleton-Merten Syndrome.Gene-Expression Analysis Identifies IGFBP2 Dysregulation in Dental Pulp Cells From Human Cleidocranial Dysplasia.

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Establishment of Singleton-Merten syndrome pulp cells: evidence of mineralization dysregulation.

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2014 nî lūn-bûn

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Establishment of Singleton-Mer ...... mineralization dysregulation.

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Establishment of Singleton-Mer ...... mineralization dysregulation.

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Establishment of Singleton-Mer ...... mineralization dysregulation.

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Establishment of Singleton-Mer ...... mineralization dysregulation.

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Establishment of Singleton-Mer ...... mineralization dysregulation.

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Establishment of Singleton-Mer ...... mineralization dysregulation.

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Establishment of Singleton-Mer ...... mineralization dysregulation.

@en

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Changming Lu

http://www.w3.org/2001/XMLSchema#string

Chun Cui

http://www.w3.org/2001/XMLSchema#string

Frank Rutsch

http://www.w3.org/2001/XMLSchema#string

Hope Amm

http://www.w3.org/2001/XMLSchema#string

Mary MacDougall

http://www.w3.org/2001/XMLSchema#string

Olga A Mamaeva

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P2860

Tooth root dentin mineralization defects in a mouse model of hypophosphatasia.

Tissue- and cell-specific alternative splicing of NFIC.

Collagens and collagen-related diseases.

The role of matrix metalloproteinases in the oral environment.

Inactivation of Tgfbr2 in Osterix-Cre expressing dental mesenchyme disrupts molar root formation.

Msx1 regulates proliferation and differentiation of mouse dental mesenchymal cells in culture.

Involvement of matrix metalloproteinases in the onset of dentin mineralization.

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57-61

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scholarly article

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10.3109/03008207.2014.923880

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55 Suppl 1

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2014-08-01T00:00:00Z

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265056085

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25158182

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Establishment of Singleton-Merten syndrome pulp cells: evidence of mineralization dysregulation.

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P2860

P2860

Establishment of Singleton-Merten syndrome pulp cells: evidence of mineralization dysregulation.

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