Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.

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Complement Activation in Liver Transplantation: Role of Donor Macrosteatosis and Implications in Delayed Graft Function.

P2860

Q55711128-D9D9B4C3-706A-429D-8E7F-B0F13195DF4C

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Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.

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http://www.wikidata.org/entity/Q46310923

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年學術文章

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name

Natural IgM initiates an infla ...... usion injury and regeneration.

@en

Natural IgM initiates an infla ...... usion injury and regeneration.

@nl

type

Item

label

Natural IgM initiates an infla ...... usion injury and regeneration.

@en

Natural IgM initiates an infla ...... usion injury and regeneration.

@nl

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Natural IgM initiates an infla ...... usion injury and regeneration.

@en

Natural IgM initiates an infla ...... usion injury and regeneration.

@nl

P1476

Natural immunoglobulin M initi ...... njury and regeneration in mice

@en

P2093

Biao Lei

http://www.w3.org/2001/XMLSchema#string

Carl Atkinson

http://www.w3.org/2001/XMLSchema#string

Fei Qiao

http://www.w3.org/2001/XMLSchema#string

Keely Marshall

http://www.w3.org/2001/XMLSchema#string

Kenneth D Chavin

http://www.w3.org/2001/XMLSchema#string

Songqing He

http://www.w3.org/2001/XMLSchema#string

Stephen Tomlinson

http://www.w3.org/2001/XMLSchema#string

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A novel role of complement: mice deficient in the fifth component of complement (C5) exhibit impaired liver regeneration.

Attenuation of skeletal muscle reperfusion injury with intravenous 12 amino acid peptides that bind to pathogenic IgM.

Pathogenic natural antibodies recognizing annexin IV are required to develop intestinal ischemia-reperfusion injury.

Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection.

Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction

C-reactive protein binds to both oxidized LDL and apoptotic cells through recognition of a common ligand: Phosphorylcholine of oxidized phospholipids

T cell-derived lymphotoxin regulates liver regeneration

B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion.

Targeting pathogenic postischemic self-recognition by natural IgM to protect against posttransplantation cardiac reperfusion injury

A complement-IL-4 regulatory circuit controls liver regeneration.

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721-735

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scholarly article

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10.1002/HEP.29512

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English

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http://www.w3.org/2001/XMLSchema#string

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http://www.w3.org/2001/XMLSchema#string

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Ali Alawieh

Junfei Jin

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2017-12-26T00:00:00Z

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28880403

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reperfusion injury

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5842100

http://www.w3.org/2001/XMLSchema#string

Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.

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P2860

P2860

Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.

description

name

type

label

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P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P921

P932