Huperzine A alleviates neuroinflammation, oxidative stress and improves cognitive function after repetitive traumatic brain injury.

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Huperzine A alleviates neuroinflammation, oxidative stress and improves cognitive function after repetitive traumatic brain injury.

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http://www.wikidata.org/entity/Q46330122

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

@zh-cn

2017年学术文章

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2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

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2017年學術文章

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name

Huperzine A alleviates neuroin ...... titive traumatic brain injury.

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Huperzine A alleviates neuroin ...... titive traumatic brain injury.

@nl

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Item

label

Huperzine A alleviates neuroin ...... titive traumatic brain injury.

@en

Huperzine A alleviates neuroin ...... titive traumatic brain injury.

@nl

prefLabel

Huperzine A alleviates neuroin ...... titive traumatic brain injury.

@en

Huperzine A alleviates neuroin ...... titive traumatic brain injury.

@nl

P1476

Huperzine A alleviates neuroin ...... titive traumatic brain injury.

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P2093

Bing Situ

http://www.w3.org/2001/XMLSchema#string

Peiying Zheng

http://www.w3.org/2001/XMLSchema#string

Xiangping Tan

http://www.w3.org/2001/XMLSchema#string

Ying Wang

http://www.w3.org/2001/XMLSchema#string

Zhengrong Mei

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P2860

Anti-inflammatory therapy in chronic disease: challenges and opportunities

Chronic neuropathologies of single and repetitive TBI: substrates of dementia?

Cognitive Impairments Induced by Concussive Mild Traumatic Brain Injury in Mouse Are Ameliorated by Treatment with Phenserine via Multiple Non-Cholinergic and Cholinergic Mechanisms

Clinical correlates in an experimental model of repetitive mild brain injury.

Axonal injury in young pediatric head trauma: a comparison study of β-amyloid precursor protein (β-APP) immunohistochemical staining in traumatic and nontraumatic deaths.

Increased Amyloid Precursor Protein and Tau Expression Manifests as Key Secondary Cell Death in Chronic Traumatic Brain Injury.

Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.

Inhibition of NR2B phosphorylation restores alterations in NMDA receptor expression and improves functional recovery following traumatic brain injury in mice

Axonal pathology in traumatic brain injury.

Human emotion experiences can be predicted on theoretical grounds: evidence from verbal labeling

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s11011-017-0075-4

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1861-1869

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scholarly article

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10.1007/S11011-017-0075-4

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2017-07-26T00:00:00Z

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28748496

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P921

neuronitis