Interaction among heme oxygenase, nuclear factor-kappaB, and transcription activating factors in cardiac hypertrophy in hypertension.
about
Heme oxygenase-1 promotes granuloma development and protects against dissemination of mycobacteria.The coordinated increased expression of biliverdin reductase and heme oxygenase-2 promotes cardiomyocyte survival: a reductase-based peptide counters β-adrenergic receptor ligand-mediated cardiac dysfunction.Tumor suppressor A20 protects against cardiac hypertrophy and fibrosis by blocking transforming growth factor-beta-activated kinase 1-dependent signalingHeme oxygenase-1 induction enhances cell survival and restores contractility to unvascularized three-dimensional adult cardiomyocyte grafts implanted in vivo.Uridine adenosine tetraphosphate-induced contraction is increased in renal but not pulmonary arteries from DOCA-salt hypertensive rats.Effects of heme oxygenase-1 upregulation on blood pressure and cardiac function in an animal model of hypertensive myocardial infarctionFeatured article: induction of heme oxygenase with hemin improves pericardial adipocyte morphology and function in obese Zucker rats by enhancing proteins of regeneration.Redox signaling in pathophysiology of hypertension.Gene transfer of inducible nitric oxide synthase affords cardioprotection by upregulating heme oxygenase-1 via a nuclear factor-{kappa}B-dependent pathway.The risk of heart failure and cardiometabolic complications in obesity may be masked by an apparent healthy status of normal blood glucose.The heme oxygenase system suppresses perirenal visceral adiposity, abates renal inflammation and ameliorates diabetic nephropathy in Zucker diabetic fatty rats.Role of heme oxygenase in inflammation, insulin-signalling, diabetes and obesity.Practical strategies for modulating foam cell formation and behavior.Molecular mechanism underlying the cerebral effect of Gly-Pro-Glu tripeptide bound to L-dopa in a Parkinson's animal model.Inflammatory cell markers as indicators of atherosclerotic renovascular disease.Nrf2 is a critical modulator of the innate immune response in a model of uveitis.Heme-arginate suppresses phospholipase C and oxidative stress in the mesenteric arterioles of mineralcorticoid-induced hypertensive rats.The heme oxygenase system attenuates pancreatic lesions and improves insulin sensitivity and glucose metabolism in deoxycorticosterone acetate hypertension.Heme arginate suppresses cardiac lesions and hypertrophy in deoxycorticosterone acetate-salt hypertension.Upregulation of the heme oxygenase system ameliorates postprandial and fasting hyperglycemia in type 2 diabetes.Heme oxygenase system enhances insulin sensitivity and glucose metabolism in streptozotocin-induced diabetes.Hemin therapy attenuates kidney injury in deoxycorticosterone acetate-salt hypertensive rats.Defensive effect of natrium diethyldithiocarbamate trihydrate (NDDCT) and lisinopril in DOCA-salt hypertension-induced vascular dementia in rats.
P2860
Q33596050-7C2710A7-1014-40C0-9904-CB6F0B1E2E86Q34421837-82103261-257C-4FF5-94D4-990B501CF86AQ34615889-41A7A386-9F24-4328-8BCF-E045518A8B5EQ34995661-9C61D01C-2CEC-4E82-BD47-E81A221C7169Q35159887-C58AE715-C485-42A5-B18F-47D829AA40B6Q36655988-0ECD60BB-DF1F-4761-B260-ED81E413932CQ37070966-0B67E874-3795-4FD1-BC25-9D12D2F65F2AQ37273310-FDFE8937-8583-4EC6-BD19-FD523A9D00BFQ37401916-012AD9E0-B1F5-4451-8B9A-F98E84955225Q37421880-4DB47B91-ADF7-4EF2-A3AE-A694DDE472B6Q37534705-244B6D38-16DE-4813-A352-0348EBAD3FDFQ37761303-C9D76C6A-EC0E-4FFC-A9DC-2D1F34D629ACQ38261037-66C07AB5-CF6C-4B3E-936C-7BD0A3B689C1Q42499967-4054BA35-E729-4815-B346-97B30B97E301Q42677522-3BE74FCE-CB44-43F0-ABAF-ECA864862F64Q43107333-3D9EAF10-69C7-4F15-ADE2-223961360D2DQ43142472-7F836FD7-0B31-4FAA-B0AA-E43BFA792759Q43253217-2544ED02-2870-4538-96F5-C24FDC55CC1DQ46017501-ADDE4294-A824-4333-9DDF-D494EC5E504AQ46125858-0537E717-6BE0-4B43-B3DE-925337489A9AQ46135112-109A82B2-29D0-4285-A4DA-EFB8283B2D91Q46179217-0E9DC1A8-CB85-4482-81C9-9101171B96D5Q48553675-8981A02C-1E35-48FA-8B54-5CF640A8D9FD
P2860
Interaction among heme oxygenase, nuclear factor-kappaB, and transcription activating factors in cardiac hypertrophy in hypertension.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@en
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@nl
type
label
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@en
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@nl
prefLabel
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@en
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@nl
P2093
P1433
P1476
Interaction among heme oxygena ...... c hypertrophy in hypertension.
@en
P2093
Ashok Jadhav
Emina Torlakovic
Joseph Fomusi Ndisang
P304
P356
10.1161/HYPERTENSIONAHA.108.114801
P407
P577
2008-09-29T00:00:00Z