CXCL16 deficiency attenuates acetaminophen-induced hepatotoxicity through decreasing hepatic oxidative stress and inflammation in mice.

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CXCL16 deficiency attenuates acetaminophen-induced hepatotoxicity through decreasing hepatic oxidative stress and inflammation in mice.

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2017 nî lūn-bûn

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CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

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CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

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CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

@en

CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

@nl

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CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

@en

CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

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P1476

CXCL16 deficiency attenuates a ...... ress and inflammation in mice.

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P2093

Anqi Xie

http://www.w3.org/2001/XMLSchema#string

Hong Wang

http://www.w3.org/2001/XMLSchema#string

Leigang Jin

http://www.w3.org/2001/XMLSchema#string

Lihua Shi

http://www.w3.org/2001/XMLSchema#string

Saisai Zhang

http://www.w3.org/2001/XMLSchema#string

Shulin Yang

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Xiaokun Li

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Xuebo Pan

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Yanna Ye

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Yihui Shao

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c-Jun N-terminal kinase modulates oxidant stress and peroxynitrite formation independent of inducible nitric oxide synthase in acetaminophen hepatotoxicity.

Hepatic response to sepsis: interaction between coagulation and inflammatory processes.

Acetaminophen and the U.S. Acute Liver Failure Study Group: lowering the risks of hepatic failure.

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A disintegrin and metalloproteinase 10-mediated cleavage and shedding regulates the cell surface expression of CXC chemokine ligand 16.

P304

541-549

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P31

scholarly article

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10.1093/ABBS/GMX040

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P433

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P478

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P577

2017-04-28T00:00:00Z

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P698

28459937

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P921

hepatotoxicity