Comparison of the myoplasmic calcium transient elicited by an action potential in intact fibres of mdx and normal mice.
about
Critical Role of Intracellular RyR1 Calcium Release Channels in Skeletal Muscle Function and DiseaseAltered nuclear dynamics in MDX myofibers.In vivo assessment of artery smooth muscle [Ca2+]i and MLCK activation in FRET-based biosensor mice.Analysis of spontaneous and nerve-evoked calcium transients in intact extraocular muscles in vitro.Eccentric exercise in aging and diseased skeletal muscle: good or bad?Excitation-contraction coupling alterations in mdx and utrophin/dystrophin double knockout mice: a comparative study.Calcium indicators and calcium signalling in skeletal muscle fibres during excitation-contraction coupling.Altered Ca(2+) signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's diseaseMitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle.The role of proteases in excitation-contraction coupling failure in muscular dystrophy.Sarcoplasmic reticulum Ca2+ permeation explored from the lumen side in mdx muscle fibers under voltage controlIntracellular calcium movements during excitation-contraction coupling in mammalian slow-twitch and fast-twitch muscle fibersIncreased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubesAbnormalities in brain structure and biochemistry associated with mdx mice measured by in vivo MRI and high resolution localized (1)H MRS.Structural and functional evaluation of branched myofibers lacking intermediate filamentsAbsence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling.Low-affinity Ca2+ indicators compared in measurements of skeletal muscle Ca2+ transients.Exercise and muscular dystrophy: implications and analysis of effects on musculoskeletal and cardiovascular systems.Disruption of action potential and calcium signaling properties in malformed myofibers from dystrophin-deficient mice.Non-invasive assessment of muscle injury in healthy and dystrophic animals with electrical impedance myography.Muscle dysfunction in a zebrafish model of Duchenne muscular dystrophy.Oxidative stress-induced dysregulation of excitation-contraction coupling contributes to muscle weakness
P2860
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P2860
Comparison of the myoplasmic calcium transient elicited by an action potential in intact fibres of mdx and normal mice.
description
2008 nî lūn-bûn
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2008年の論文
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2008年学术文章
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2008年学术文章
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2008年学术文章
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2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
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2008年學術文章
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2008年學術文章
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name
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@en
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@nl
type
label
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@en
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@nl
prefLabel
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@en
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@nl
P2093
P2860
P1476
Comparison of the myoplasmic c ...... fibres of mdx and normal mice.
@en
P2093
Stephen Hollingworth
Stephen M Baylor
Ulrike Zeiger
P2860
P304
P356
10.1113/JPHYSIOL.2008.160507
P407
P577
2008-09-04T00:00:00Z