Muscle protein synthesis, mTORC1/MAPK/Hippo signaling, and capillary density are altered by blocking of myostatin and activins.
about
YAP-Mediated Mechanotransduction in Skeletal MuscleDifferential muscle hypertrophy is associated with satellite cell numbers and Akt pathway activation following activin type IIB receptor inhibition in Mtm1 p.R69C miceSkeletal muscle tissue engineering: methods to form skeletal myotubes and their applications.Upregulation of activin-B and follistatin in pulmonary fibrosis - a translational study using human biopsies and a specific inhibitor in mouse fibrosis models.Signals of Ezh2, Src, and Akt Involve in myostatin-Pax7 pathways regulating the myogenic fate determination during the sheep myoblast proliferation and differentiationIntestinal Commitment and Maturation of Human Pluripotent Stem Cells Is Independent of Exogenous FGF4 and R-spondin1Treatment with soluble activin type IIB-receptor improves bone mass and strength in a mouse model of Duchenne muscular dystrophy.Inhibition of myostatin signaling through Notch activation following acute resistance exercise.Smad3 induces atrogin-1, inhibits mTOR and protein synthesis, and promotes muscle atrophy in vivo.Systemic blockade of ACVR2B ligands prevents chemotherapy-induced muscle wasting by restoring muscle protein synthesis without affecting oxidative capacity or atrogenes.New roles for Smad signaling and phosphatidic acid in the regulation of skeletal muscle mass.Hippo Pathway and Skeletal Muscle Mass Regulation in Mammals: A Controversial Relationship.Gremlin-1 associates with fibrillin microfibrils in vivo and regulates mesothelioma cell survival through transcription factor slug.Application of a nonradioactive method of measuring protein synthesis in industrially relevant Chinese hamster ovary cells.Inhibition of Activin Signaling Slows Progression of Polycystic Kidney Disease.Soluble activin type IIB receptor improves fracture healing in a closed tibial fracture mouse model.Fluid shear stress-induced TGF-β/ALK5 signaling in renal epithelial cells is modulated by MEK1/2.Gremlin-1 is a key regulator of the invasive cell phenotype in mesothelioma.Prevention of chemotherapy-induced cachexia by ACVR2B ligand blocking has different effects on heart and skeletal muscle.Exercise restores decreased physical activity levels and increases markers of autophagy and oxidative capacity in myostatin/activin-blocked mdx mice.Treating cachexia using soluble ACVR2B improves survival, alters mTOR localization, and attenuates liver and spleen responses.
P2860
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P2860
Muscle protein synthesis, mTORC1/MAPK/Hippo signaling, and capillary density are altered by blocking of myostatin and activins.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh-hant
name
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@en
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@nl
type
label
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@en
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@nl
prefLabel
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@en
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@nl
P2093
P2860
P50
P1476
Muscle protein synthesis, mTOR ...... ing of myostatin and activins.
@en
P2093
Arja Pasternack
Bernardo M Oliveira
Hongqiang Ma
Olli Ritvos
Philippe Pierre
Willem M H Hoogaars
P2860
P304
P356
10.1152/AJPENDO.00389.2012
P577
2012-10-31T00:00:00Z