Social interaction deficits caused by chronic phencyclidine administration are reversed by oxytocin. - Wikidata - awgldk - TriplyDB

Social interaction deficits caused by chronic phencyclidine administration are reversed by oxytocin.

Social interaction deficits caused by chronic phencyclidine administration are reversed by oxytocin.

http://www.wikidata.org/entity/Q46411318

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From ultrasocial to antisocial: a role for oxytocin in the acute reinforcing effects and long-term adverse consequences of drug use?Oxytocin: the great facilitator of life.Oxytocin in the socioemotional brain: implications for psychiatric disorders Prenatal stress generates deficits in rat social behavior: Reversal by oxytocin Social Neuroscience: Progress and Implications for Mental Health Heterozygous Disruption of Autism susceptibility candidate 2 Causes Impaired Emotional Control and Cognitive Memory Impaired limbic cortico-striatal structure and sustained visual attention in a rodent model of schizophrenia.A Role for Oxytocin in the Etiology and Treatment of Schizophrenia.Postmortem brain: an underutilized substrate for studying severe mental illness The effects of oxytocin and its analog, carbetocin, on genetic deficits in sensorimotor gating.Oxytocin in schizophrenia: a review of evidence for its therapeutic effects.Iptakalim Preferentially Decreases Nicotine-induced Hyperlocomotion in Phencyclidine-sensitized Rats: A Potential Dual Action against Nicotine Addiction and Psychosis.Animal models of schizophrenia.Oxytocin, dopamine, and the amygdala: a neurofunctional model of social cognitive deficits in schizophrenia Prenatal stress: role in psychotic and depressive diseases.The mechanism of life-threatening water imbalance in schizophrenia and its relationship to the underlying psychiatric illness.The evolution of drug development in schizophrenia: past issues and future opportunities.Using the MATRICS to guide development of a preclinical cognitive test battery for research in schizophrenia.Effects of bisphenol-A and other endocrine disruptors compared with abnormalities of schizophrenia: an endocrine-disruption theory of schizophrenia.Oxytocin reduces amygdala activity, increases social interactions, and reduces anxiety-like behavior irrespective of NMDAR antagonism Peripubertal stress-induced behavioral changes are associated with altered expression of genes involved in excitation and inhibition in the amygdala.Neurobiology of sociability.Pharmacological treatment of schizophrenia: a critical review of the pharmacology and clinical effects of current and future therapeutic agents.Peripheral oxytocin is associated with reduced symptom severity in schizophrenia Antiaggressive activity of central oxytocin in male rats.Overexpression of oxytocin receptors in the hypothalamic PVN increases baroreceptor reflex sensitivity and buffers BP variability in conscious rats.Vardenafil Enhances Oxytocin Expression in the Paraventricular Nucleus without Sexual Stimulation.mGluR2/3 agonist LY379268 rescues NMDA and GABAA receptor level deficits induced in a two-hit mouse model of schizophrenia.Association between oxytocin receptor gene polymorphisms and self-rated 'empathic concern' in schizophrenia.Hydration state controls stress responsiveness and social behavior.Peripherally administered oxytocin modulates latent inhibition in a manner consistent with antipsychotic drugs Oxytocin as a potential therapeutic target for schizophrenia and other neuropsychiatric conditions.Dopamine in socioecological and evolutionary perspectives: implications for psychiatric disorders.Social interaction and social withdrawal in rodents as readouts for investigating the negative symptoms of schizophrenia.Effects of hallucinogenic agents mescaline and phencyclidine on zebrafish behavior and physiology.The Effect of Oxytocin on Social and Non-Social Behaviour and Striatal Protein Expression in C57BL/6N Mice.Stem cell therapy: social recognition recovery in a FASD model.Helping oxytocin deliver: considerations in the development of oxytocin-based therapeutics for brain disorders.Vasopressin and oxytocin in control of the cardiovascular system Effects of Subchronic Phencyclidine (PCP) Treatment on Social Behaviors, and Operant Discrimination and Reversal Learning in C57BL/6J Mice.

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Social interaction deficits caused by chronic phencyclidine administration are reversed by oxytocin.

http://www.wikidata.org/entity/Q46411318

description

2005 nî lūn-bûn

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2005年の論文

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Social interaction deficits ca ...... tion are reversed by oxytocin.

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Social interaction deficits ca ...... tion are reversed by oxytocin.

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Social interaction deficits ca ...... tion are reversed by oxytocin.

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Social interaction deficits ca ...... tion are reversed by oxytocin.

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Social interaction deficits ca ...... tion are reversed by oxytocin.

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Dana L Brady

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Daniel M Dorsa

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James I Koenig

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Paul R Lee

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Robert A Shapiro

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P2860

Conditional calcineurin knockout mice exhibit multiple abnormal behaviors related to schizophrenia

Subchronic phencyclidine administration alters central vasopressin receptor binding and social interaction in the rat

Expanded characterization of the social interaction abnormalities in mice lacking Dvl1

Mice with reduced NMDA receptor expression display behaviors related to schizophrenia

Social interaction and sensorimotor gating abnormalities in mice lacking Dvl1

Subanesthetic effects of the noncompetitive NMDA antagonist, ketamine, in humans. Psychotomimetic, perceptual, cognitive, and neuroendocrine responses

Recent advances in the phencyclidine model of schizophrenia

Clozapine, but not haloperidol, reverses social behavior deficit in mice during withdrawal from chronic phencyclidine treatment.

D-Cycloserine added to risperidone in patients with primary negative symptoms of schizophrenia.

To model a psychiatric disorder in animals: schizophrenia as a reality test.

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