Direct effects of interleukin-13 on epithelial cells cause airway hyperreactivity and mucus overproduction in asthma. - Wikidata - awgldk - TriplyDB

Direct effects of interleukin-13 on epithelial cells cause airway hyperreactivity and mucus overproduction in asthma.

Direct effects of interleukin-13 on epithelial cells cause airway hyperreactivity and mucus overproduction in asthma.

http://www.wikidata.org/entity/Q46413344

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Linoleic acid metabolite drives severe asthma by causing airway epithelial injury Differential enzymatic activity of common haplotypic versions of the human acidic Mammalian chitinase protein The adipocyte fatty acid-binding protein aP2 is required in allergic airway inflammation A distinctive alveolar macrophage activation state induced by cigarette smoking Beta2-adrenoceptor signaling is required for the development of an asthma phenotype in a murine model The epithelial anion transporter pendrin is induced by allergy and rhinovirus infection, regulates airway surface liquid, and increases airway reactivity and inflammation in an asthma model Inducible expression of the proallergic cytokine thymic stromal lymphopoietin in airway epithelial cells is controlled by NFkappaB SPDEF regulates goblet cell hyperplasia in the airway epithelium IL-13 and epidermal growth factor receptor have critical but distinct roles in epithelial cell mucin production Aspergillus antigen induces robust Th2 cytokine production, inflammation, airway hyperreactivity and fibrosis in the absence of MCP-1 or CCR2 The effect of IL-13 and IL-13R130Q, a naturally occurring IL-13 polymorphism, on the gene expression of human airway smooth muscle cells Antigen-sensitized CD4+CD62Llow memory/effector T helper 2 cells can induce airway hyperresponsiveness in an antigen free setting STAT6 and PARP Family Members in the Development of T Cell-dependent Allergic Inflammation Stabilization of high-risk plaques The origin and role of innate lymphoid cells in the lung Dendritic cells in lung immunopathology Epithelial Sodium and Chloride Channels and Asthma Host responses in tissue repair and fibrosis IL-4 and IL-13 signaling in allergic airway disease Strategic applications of gene expression: from drug discovery/development to bedside The cell biology of asthma IL-13-induced airway mucus production is attenuated by MAPK13 inhibition.Using Periostin as a Biomarker in the Treatment of Asthma Gene expression profiles reveal increased mClca3 (Gob5) expression and mucin production in a murine model of asbestos-induced fibrogenesis Role of infection in the development and exacerbation of asthma SPDEF is required for mouse pulmonary goblet cell differentiation and regulates a network of genes associated with mucus production Development of pre-clinical models for evaluating the therapeutic potential of candidate siRNA targeting STAT6 1,8-Cineol Reduces Mucus-Production in a Novel Human Ex Vivo Model of Late Rhinosinusitis The role of CLCA proteins in inflammatory airway disease Multiwalled Carbon Nanotube Functionalization with High Molecular Weight Hyaluronan Significantly Reduces Pulmonary Injury Mechanisms of fibrosis: therapeutic translation for fibrotic disease IL-13-induced proliferation of airway epithelial cells: mediation by intracellular growth factor mobilization and ADAM17 Genome-wide profiling identifies epithelial cell genes associated with asthma and with treatment response to corticosteroids The phosphatase CD148 promotes airway hyperresponsiveness through SRC family kinases.B cell antigen presentation promotes Th2 responses and immunopathology during chronic allergic lung disease IL-13 expression by blood T cells and not eosinophils is increased in asthma compared to non-asthmatic eosinophilic bronchitis.Airway hyperresponsiveness is associated with airway remodeling but not inflammation in aging Cav1-/- mice.Allergic asthma is distinguished by sensitivity of allergen-specific CD4+ T cells and airway structural cells to type 2 inflammation.Intestinal epithelial cell secretion of RELM-beta protects against gastrointestinal worm infection.Somatic extracts of Marshallagia marshalli downregulate the Th2 associated immune responses in ovalbumin-induced airway inflammation in BALB/c mice.

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Direct effects of interleukin-13 on epithelial cells cause airway hyperreactivity and mucus overproduction in asthma.

http://www.wikidata.org/entity/Q46413344

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David J Erle

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Dean Sheppard

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Douglas A Kuperman

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Grace H Chang

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Gregory M Dolganov

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