Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans.
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Host response to Candida albicans bloodstream infection and sepsisEarly-expressed chemokines predict kidney immunopathology in experimental disseminated Candida albicans infectionsRecombinant Human IFNα-2b Response Promotes Vaginal Epithelial Cells Defense against Candida albicans.A novel role for the NLRC4 inflammasome in mucosal defenses against the fungal pathogen Candida albicansGranulocyte colony-stimulating factor protects mice during respiratory virus infections.Cytosolic phospholipase A(2)α and eicosanoids regulate expression of genes in macrophages involved in host defense and inflammation.Systemic responses during local viral infections: type I IFNs sound the alarm.Acquisition of antigen-presenting functions by neutrophils isolated from mice with chronic colitis.Abolishing Cell Wall Glycosylphosphatidylinositol-Anchored Proteins in Candida albicans Enhances Recognition by Host Dectin-1.Langerhans Cells Suppress CD49a+ NK Cell-Mediated Skin Inflammation.MCPIP1 Endoribonuclease Activity Negatively Regulates Interleukin-17-Mediated Signaling and Inflammation.The Role of Dectin-2 for Host Defense Against Disseminated CandidiasisMnn10 Maintains Pathogenicity in Candida albicans by Extending α-1,6-Mannose Backbone to Evade Host Dectin-1 Mediated Antifungal Immunity.Bst1 is required for Candida albicans infecting host via facilitating cell wall anchorage of Glycosylphosphatidyl inositol anchored proteins.Role of Dectin-2 for host defense against systemic infection with Candida glabrata.Innate and adaptive antifungal immune responses: partners on an equal footing.Antifungal Immunological Defenses in Newborns.The Goldilocks model of immune symbiosis with Mycobacteria and Candida colonizers.Antibody-Independent Function of Human B Cells Contributes to Antifungal T Cell Responses.Macrophages protect Talaromyces marneffei conidia from myeloperoxidase-dependent neutrophil fungicidal activity during infection establishment in vivo.Involvement of the capsular GalXM-induced IL-17 cytokine in the control of Cryptococcus neoformans infection
P2860
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P2860
Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
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2008年學術文章
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name
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@en
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@nl
type
label
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@en
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@nl
prefLabel
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@en
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@nl
P2093
P2860
P1476
Mice lacking both G-CSF and IL ...... ense against Candida albicans.
@en
P2093
Ashley R Dunn
Cathy Quilici
Dianne Grail
Hui-Hua Zhang
Sunanda Basu
P2860
P356
10.1080/08977190801987513
P577
2008-02-01T00:00:00Z