Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans. - Wikidata - awgldk - TriplyDB

Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans.

Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans.

http://www.wikidata.org/entity/Q46434311

about

Host response to Candida albicans bloodstream infection and sepsis Early-expressed chemokines predict kidney immunopathology in experimental disseminated Candida albicans infections Recombinant Human IFNα-2b Response Promotes Vaginal Epithelial Cells Defense against Candida albicans.A novel role for the NLRC4 inflammasome in mucosal defenses against the fungal pathogen Candida albicans Granulocyte colony-stimulating factor protects mice during respiratory virus infections.Cytosolic phospholipase A(2)α and eicosanoids regulate expression of genes in macrophages involved in host defense and inflammation.Systemic responses during local viral infections: type I IFNs sound the alarm.Acquisition of antigen-presenting functions by neutrophils isolated from mice with chronic colitis.Abolishing Cell Wall Glycosylphosphatidylinositol-Anchored Proteins in Candida albicans Enhances Recognition by Host Dectin-1.Langerhans Cells Suppress CD49a+ NK Cell-Mediated Skin Inflammation.MCPIP1 Endoribonuclease Activity Negatively Regulates Interleukin-17-Mediated Signaling and Inflammation.The Role of Dectin-2 for Host Defense Against Disseminated Candidiasis Mnn10 Maintains Pathogenicity in Candida albicans by Extending α-1,6-Mannose Backbone to Evade Host Dectin-1 Mediated Antifungal Immunity.Bst1 is required for Candida albicans infecting host via facilitating cell wall anchorage of Glycosylphosphatidyl inositol anchored proteins.Role of Dectin-2 for host defense against systemic infection with Candida glabrata.Innate and adaptive antifungal immune responses: partners on an equal footing.Antifungal Immunological Defenses in Newborns.The Goldilocks model of immune symbiosis with Mycobacteria and Candida colonizers.Antibody-Independent Function of Human B Cells Contributes to Antifungal T Cell Responses.Macrophages protect Talaromyces marneffei conidia from myeloperoxidase-dependent neutrophil fungicidal activity during infection establishment in vivo.Involvement of the capsular GalXM-induced IL-17 cytokine in the control of Cryptococcus neoformans infection

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Mice lacking both G-CSF and IL-6 are more susceptible to Candida albicans infection: critical role of neutrophils in defense against Candida albicans.

http://www.wikidata.org/entity/Q46434311

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

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2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh-hant

name

Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@en

Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@nl

type

label

Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@en

Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@nl

prefLabel

Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@en

Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@nl

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08977190801987513

P1433

Growth Factors Journal

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Mice lacking both G-CSF and IL ...... ense against Candida albicans.

@en

P2093

Ashley R Dunn

http://www.w3.org/2001/XMLSchema#string

Cathy Quilici

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Dianne Grail

http://www.w3.org/2001/XMLSchema#string

Hui-Hua Zhang

http://www.w3.org/2001/XMLSchema#string

Sunanda Basu

http://www.w3.org/2001/XMLSchema#string

P2860

Infections in bone marrow transplant recipients.

Fungal infections after bone marrow transplant.

Immunity to Candida albicans: Th1, Th2 cells and beyond.

Protective role of interleukin-6 during Yersinia enterocolitica infection is mediated through the modulation of inflammatory cytokines

The two faces of IL-6 on Th1/Th2 differentiation.

Immunity to fungal infections.

Interleukin 6 plays a key role in the development of antigen-induced arthritis.

Impaired neutrophil response and CD4+ T helper cell 1 development in interleukin 6-deficient mice infected with Candida albicans

Interleukin (IL)-6 directs the differentiation of IL-4-producing CD4+ T cells

Colony-forming cells with high proliferative potential (HPP-CFC).

P304

23-34

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scholarly article

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10.1080/08977190801987513

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1

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P478

26

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P577

2008-02-01T00:00:00Z

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P698

18365876

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