Thrombin increases permeability only in venules exposed to inflammatory conditions.
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Tonic regulation of vascular permeabilityEnhanced permeability responses to inflammation in streptozotocin-induced diabetic rat venules: Rho-mediated alterations of actin cytoskeleton and VE-cadherinVascular remodeling alters adhesion protein and cytoskeleton reactions to inflammatory stimuli resulting in enhanced permeability increases in rat venules.Vascular permeability modulation at the cell, microvessel, or whole organ level: towards closing gaps in our knowledge.Microperfusion Technique to Investigate Regulation of Microvessel Permeability in Rat Mesentery.Lymphatic vascular integrity is disrupted in type 2 diabetes due to impaired nitric oxide signalling.Dynamics of neutrophil extravasation and vascular permeability are uncoupled during aseptic cutaneous wounding.The role of atrial natriuretic peptide to attenuate inflammation in a mouse skin wound and individually perfused rat mesenteric microvessels.Mind the gap: mechanisms regulating the endothelial barrier.Thrombin-induced endothelial barrier disruption in intact microvessels: role of RhoA/Rho kinase-myosin phosphatase axis.Sialic acids regulate microvessel permeability, revealed by novel in vivo studies of endothelial glycocalyx structure and functionRole of CL-100, a dual specificity phosphatase, in thrombin-induced endothelial cell activation.Oncotic pressures opposing filtration across non-fenestrated rat microvessels.Permeability measurements in an individually perfused capillary: the 'squid axon' of the microcirculation (1974).Epac/Rap1 pathway regulates microvascular hyperpermeability induced by PAF in rat mesentery.Thrombin enhances the barrier function of rat microvascular endothelium in a PAR-1-dependent manner.Shear stress regulates HUVEC hydraulic conductivity by occludin phosphorylation.Histamine causes endothelial barrier disruption via Ca-mediated RhoA activation and tension at adherens junctions
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P2860
Thrombin increases permeability only in venules exposed to inflammatory conditions.
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2003 nî lūn-bûn
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2003年の論文
@ja
2003年学术文章
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2003年学术文章
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2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
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2003年學術文章
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2003年學術文章
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name
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@en
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@nl
type
label
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@en
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@nl
prefLabel
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@en
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@nl
P2093
P2860
P1476
Thrombin increases permeability only in venules exposed to inflammatory conditions.
@en
P2093
P2860
P304
P356
10.1152/AJPHEART.00262.2003
P577
2003-07-31T00:00:00Z