Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
about
Repetitive pertussis toxin promotes development of regulatory T cells and prevents central nervous system autoimmune diseaseIdentification of soluble TREM-2 in the cerebrospinal fluid and its association with multiple sclerosis and CNS inflammationDouble Roles of Macrophages in Human Neuroimmune Diseases and Their Animal ModelsCD8(+) T-Cells as Immune Regulators of Multiple SclerosisKynurenines and Multiple Sclerosis: The Dialogue between the Immune System and the Central Nervous SystemRemyelination therapy for multiple sclerosisImmune modulation of stem cells and regenerationFunction of microglia and macrophages in secondary damage after spinal cord injuryM1 and M2 immune activation in Parkinson's Disease: Foe and ally?Therapeutic effects of glatiramer acetate and grafted CD115⁺ monocytes in a mouse model of Alzheimer's disease.Infiltrating blood-derived macrophages are vital cells playing an anti-inflammatory role in recovery from spinal cord injury in miceGlatiramer acetate in the treatment of multiple sclerosis: emerging concepts regarding its mechanism of actionRecombinant adeno-associated viral (rAAV) vectors mediate efficient gene transduction in cultured neonatal and adult microgliaLaquinimod, a quinoline-3-carboxamide, induces type II myeloid cells that modulate central nervous system autoimmunityA one year follow-up study of natural killer and dendritic cells activities in multiple sclerosis patients receiving glatiramer acetate (GA)Glatiramer acetate (copaxone) modulates platelet activation and inhibits thrombin-induced calcium influx: possible role of copaxone in targeting platelets during autoimmune neuroinflammationTherapeutic decisions in multiple sclerosis: moving beyond efficacyType II Activation of Macrophages and Microglia by Immune Complexes Enhances Th17 Biasing in an IL-6-Independent MannerGene expression studies of a human monocyte cell line identify dissimilarities between differently manufactured glatiramoidsMacrophage-specific chemokines induced via innate immunity by amino acid copolymers and their role in EAEIdentification of Orch3, a locus controlling dominant resistance to autoimmune orchitis, as kinesin family member 1CRett syndrome and other autism spectrum disorders--brain diseases of immune malfunction?Trichuris suis induces human non-classical patrolling monocytes via the mannose receptor and PKC: implications for multiple sclerosis.Continuous retinoic acid induces the differentiation of mature regulatory monocytes but fails to induce regulatory dendritic cellsInhibitory role for GABA in autoimmune inflammation.T-cell based immunotherapy in experimental autoimmune encephalomyelitis and multiple sclerosis.Enhanced disease reduction using clozapine, an atypical antipsychotic agent, and glatiramer acetate combination therapy in experimental autoimmune encephalomyelitisPathologic heterogeneity persists in early active multiple sclerosis lesionsOnce-daily glatiramer acetate decreases magnetic resonance imaging disease activity in Japanese patients with relapsing-remitting multiple sclerosis.The post sepsis-induced expansion and enhanced function of regulatory T cells create an environment to potentiate tumor growth.Wound administration of M2-polarized macrophages does not improve murine cutaneous healing responses.Involvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis.Immune regulation of multiple sclerosis by CD8+ T cellsTreatment with the antipsychotic agent, risperidone, reduces disease severity in experimental autoimmune encephalomyelitis.Neuroprotective effect of combination therapy of glatiramer acetate and epigallocatechin-3-gallate in neuroinflammation.Developmental maturation of innate immune cell function correlates with susceptibility to central nervous system autoimmunity.Neuroantigen-specific autoregulatory CD8+ T cells inhibit autoimmune demyelination through modulation of dendritic cell function.Inhibition of experimental autoimmune uveitis by amino acid copolymersCentral nervous system (CNS)-resident natural killer cells suppress Th17 responses and CNS autoimmune pathology.Immunomodulation of antigen presenting cells promotes natural regulatory T cells that prevent autoimmune diabetes in NOD mice.
P2860
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P2860
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@en
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@nl
type
label
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@en
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@nl
prefLabel
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@en
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@nl
P2093
P2860
P356
P1433
P1476
Type II monocytes modulate T cell-mediated central nervous system autoimmune disease.
@en
P2093
Cynthia D Rundle
Juan C Patarroyo
Lawrence Steinman
Martin S Weber
Raymond A Sobel
Sawsan Youssef
Scott S Zamvil
Shannon E Dunn
Thomas Prod'homme
P2860
P2888
P304
P356
10.1038/NM1620
P407
P50
P577
2007-08-05T00:00:00Z