Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse.
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Role of the MPTP in conditioning the heart - translatability and mechanismThe GSK-3 family as therapeutic target for myocardial diseasesPostconditioning signalling in the heart: mechanisms and translatabilityIntermittent losartan administration triggers cardiac post-conditioning in isolated rat hearts: role of BK2 receptorsSelective blockade of protein kinase B protects the rat and human myocardium against ischaemic injuryBerbamine postconditioning protects the heart from ischemia/reperfusion injury through modulation of autophagyCardioprotection leads to novel changes in the mitochondrial proteome.Type 2 diabetic obese db/db mice are refractory to myocardial ischaemic post-conditioning in vivo: potential role for Hsp20, F1-ATPase δ and Echs1Constitutive glycogen synthase kinase-3alpha/beta activity protects against chronic beta-adrenergic remodelling of the heart.Myocardial stress remodelling after regional infarction is independent of glycogen synthase kinase-3 inactivation.Post-ischemic conditioning in the rat retina is dependent upon ischemia duration and is not additive with ischemic pre-conditioningMitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis.Redox signaling in cardiac myocytes.Inhibition of glycogen synthase kinase 3 beta ameliorates liver ischemia reperfusion injury by way of an interleukin-10-mediated immune regulatory mechanism.Pharmacological postconditioning effect of muramyl dipeptide is mediated through RIP2 and TAK1Isoflurane postconditioning protects against reperfusion injury by preventing mitochondrial permeability transition by an endothelial nitric oxide synthase-dependent mechanismTransgenic over expression of ectonucleotide triphosphate diphosphohydrolase-1 protects against murine myocardial ischemic injury.G Protein-Coupled Estrogen Receptor 1 Mediates Acute Estrogen-Induced Cardioprotection via MEK/ERK/GSK-3β Pathway after Ischemia/Reperfusion.Phosphorylation of GSK-3β mediates intralipid-induced cardioprotection against ischemia/reperfusion injury.Cardioprotection and altered mitochondrial adenine nucleotide transportRemote Liver Ischemic Preconditioning Protects against Sudden Cardiac Death via an ERK/GSK-3β-Dependent Mechanism.Aldose reductase modulates cardiac glycogen synthase kinase-3β phosphorylation during ischemia-reperfusion.Inhibition of glycogen synthase kinase 3beta prevents peroxide-induced collapse of mitochondrial membrane potential in rat ventricular myocytes.Nuclear and mitochondrial signalling Akts in cardiomyocytesDoes inhibition of glycogen synthase kinase protect in mice?Protective ischaemia in patients: preconditioning and postconditioning.Role of glycogen synthase kinase-3beta in cardioprotection.Akt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.Insulin suppresses ischemic preconditioning-mediated cardioprotection through Akt-dependent mechanisms.Inhibition of mitochondrial membrane permeability as a putative pharmacological target for cardioprotection.Role of the gap junction in ischemic preconditioning in the heart.Signal transduction to the permeability transition pore.Postconditioning and protection from reperfusion injury: where do we stand? Position paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology.Mechanism of cardioprotection by early ischemic preconditioningDiabetic inhibition of preconditioning- and postconditioning-mediated myocardial protection against ischemia/reperfusion injury.Role of the Wnt-Frizzled system in cardiac pathophysiology: a rapidly developing, poorly understood area with enormous potential.Redox balance and cardioprotection.Mitochondrial channels: ion fluxes and more.The cGMP/PKG pathway as a common mediator of cardioprotection: translatability and mechanism.Treatment of Myocardial Ischemia/Reperfusion Injury by Ischemic and Pharmacological Postconditioning.
P2860
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P2860
Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@en
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@nl
type
label
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@en
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@nl
prefLabel
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@en
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@nl
P2093
P50
P1433
P1476
Glycogen synthase kinase-3 ina ...... postconditioning in the mouse.
@en
P2093
Derek M Yellon
Ian G Webb
James E Clark
Sebastien Jacquet
Tetsuji Miura
Yasuhiro Nishino
P304
P356
10.1161/CIRCRESAHA.107.169953
P577
2008-06-26T00:00:00Z