A diabetes-like environment increases malformation rate and diminishes prostaglandin E(2) in rat embryos: reversal by administration of vitamin E and folic acid.
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Modeling anterior development in mice: diet as modulator of risk for neural tube defectsMaternal diabetes alters transcriptional programs in the developing embryo.Diabetic embryopathy: a role for the epigenome?New concepts in diabetic embryopathyThe status of diabetic embryopathy.Lack of periconceptional vitamins or supplements that contain folic acid and diabetes mellitus-associated birth defects.Understanding diabetic teratogenesis: where are we now and where are we going?Oxidative stress in pregnancy and fertility pathologies.Effect of maternal diabetes on the embryo, fetus, and children: congenital anomalies, genetic and epigenetic changes and developmental outcomes.Safflower and olive oil dietary treatments rescue aberrant embryonic arachidonic acid and nitric oxide metabolism and prevent diabetic embryopathy in rats.Developmental toxicity of glyceryl trinitrate in quail embryos.
P2860
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P2860
A diabetes-like environment increases malformation rate and diminishes prostaglandin E(2) in rat embryos: reversal by administration of vitamin E and folic acid.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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name
A diabetes-like environment increases malformation rate and diminishes prostaglandin E
@nl
A diabetes-like environment in ...... n of vitamin E and folic acid.
@en
type
label
A diabetes-like environment increases malformation rate and diminishes prostaglandin E
@nl
A diabetes-like environment in ...... n of vitamin E and folic acid.
@en
prefLabel
A diabetes-like environment increases malformation rate and diminishes prostaglandin E
@nl
A diabetes-like environment in ...... n of vitamin E and folic acid.
@en
P2860
P356
P1476
A diabetes-like environment in ...... n of vitamin E and folic acid.
@en
P2093
Parri Wentzel
Ulf J Eriksson
P2860
P304
P356
10.1002/BDRA.20145
P577
2005-07-01T00:00:00Z