Abeta25-35 alters Akt activity, resulting in Bad translocation and mitochondrial dysfunction in cerebrovascular endothelial cells.
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Anthraquinone-2-sulfonic acid (AQ2S) is a novel neurotherapeutic agentExtracellular signal-regulated kinase activation by Neisseria gonorrhoeae downregulates epithelial cell proapoptotic proteins Bad and Bim.Blockade of Tau hyperphosphorylation and Aβ₁₋₄₂ generation by the aminotetrahydrofuran derivative ANAVEX2-73, a mixed muscarinic and σ₁ receptor agonist, in a nontransgenic mouse model of Alzheimer's disease.Inhibition of HSP90-dependent telomerase activity in amyloid β-induced apoptosis of cerebral endothelial cells.
P2860
Abeta25-35 alters Akt activity, resulting in Bad translocation and mitochondrial dysfunction in cerebrovascular endothelial cells.
description
2005 nî lūn-bûn
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name
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@en
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@nl
type
label
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@en
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@nl
prefLabel
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@en
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@nl
P2860
P50
P356
P1476
Abeta25-35 alters Akt activity ...... brovascular endothelial cells.
@en
P2093
P2860
P304
P356
10.1038/SJ.JCBFM.9600139
P577
2005-11-01T00:00:00Z