Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
about
Human malarial disease: a consequence of inflammatory cytokine releasePfHRP2 measures schizogony, not mechanical blockageDysfunction of respiratory muscles in critically ill patients on the intensive care unitCerium oxide nanoparticle treatment ameliorates peritonitis-induced diaphragm dysfunctionLipopolysaccharide-induced weakness in the preterm diaphragm is associated with mitochondrial electron transport chain dysfunction and oxidative stress.Energy crisis: the role of oxidative phosphorylation in acute inflammation and sepsisAutophagy and skeletal muscles in sepsisRecently published papers: treating sepsis, measuring troponin and managing the obeseThe Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.Skeletal muscle dysfunction is associated with derangements in mitochondrial bioenergetics (but not UCP3) in a rodent model of sepsis.Update in critical care 2005.Bench-to-bedside review: potential strategies to protect or reverse mitochondrial dysfunction in sepsis-induced organ failure.Mitochondrial mechanisms of sepsis-induced organ failure.Diaphragm weakness in mechanically ventilated critically ill patients.MitoQ administration prevents endotoxin-induced cardiac dysfunction.Recovery of diaphragm function following mechanical ventilation in a rodent model.SIDS-CDF Hypothesis Revisited: Cause vs. Contributing Factors.Sepsis-induced myopathy.Exogenous cytochrome C restores myocardial cytochrome oxidase activity into the late phase of sepsis.Caffeine restores myocardial cytochrome oxidase activity and improves cardiac function during sepsis.Mitochondrial DNA and inflammatory diseases.Measurement of Mitochondrial Respiration and Motility in Acute Care: Sepsis, Trauma, and Poisoning.Hydrogen sulfide prevents diaphragm weakness in cecal ligation puncture-induced sepsis by preservation of mitochondrial function.Organ protective mechanisms common to extremes of physiology: a window through hibernation biology.Whey Peptide-Based Formulas With ω-3 Fatty Acids Are Protective in Lipopolysaccharide-Mediated Sepsis.Mitochondrial function in sepsis.Respiratory failure during critical illness: are mitochondria to blame?Reduced DHPRα1S and RyR1 expression levels are associated with diaphragm contractile dysfunction during sepsis.The beneficial effects of melatonin against heart mitochondrial impairment during sepsis: inhibition of iNOS and preservation of nNOS.Effects of 47C allele (rs4880) of the SOD2 gene in the production of intracellular reactive species in peripheral blood mononuclear cells with and without lipopolysaccharides induction.Diaphragm Dysfunction in Critical Illness.Glutamine restores myocardial cytochrome C oxidase activity and improves cardiac function during experimental sepsis.Diaphragm and cardiac mitochondrial creatine kinases are impaired in sepsis.
P2860
Q21034098-7EB2F5F1-756C-4175-B18F-44038C9D2902Q25256482-FDA92BC9-A63E-48E9-84A1-0C9BB50802C7Q26749263-7136ECED-EF19-4C69-9549-5EABE2D44FA2Q28390788-7FA1FD55-3C21-4880-B5E9-D235FD20B884Q31138969-0C247401-F688-44B0-B560-2CFF518EAA88Q34102154-C92EDD36-5649-4850-A607-329A5E384164Q34442945-640E1463-DF4B-47E6-B28A-A31462DF2EAEQ34480823-AC337D85-289D-44AB-A613-B558EE36FAAAQ34483183-A8391F94-F62F-4539-A4F8-444E4A58A79FQ35572934-5845440A-AF5C-4885-A305-C50F6B2494F4Q36442993-CFB1159E-4072-4541-A0D5-E69726CCDA58Q36585950-1DF99D93-6FF1-448E-989A-D8DCC7B2A7E8Q37175448-AA08FFE6-1E32-4B73-A6E9-17F4EAC5B2BCQ37343222-9877E3BC-B297-4A40-AE72-A9BD1DAC4322Q37391879-5F6FAF42-298E-4456-828D-4DFC8D96C2EDQ37521154-6A34A533-7A53-437F-BFDC-0247B32E995BQ37587218-DFEE8546-E25A-41EF-9FA2-1B517C308D47Q37666348-5A16884E-6C0A-419C-B7C6-BB7DAB9C419CQ37673307-A8900D70-7C80-4E67-9C44-582E8E0F1BB0Q37673315-06117550-CEDC-4A2C-81A0-B00CB5DA255BQ37898562-FA5410F7-E9E6-4A2C-A17C-60D4119C4768Q40604767-0885F204-9291-4888-B40C-C8BC69A7C87FQ41364237-4B2E5D1D-5E2A-423D-A771-B509A9E51D07Q43674494-0EA13BBA-C508-4D92-BEE7-BA3A32DA4FB6Q43735700-B9FADECF-1BA1-41E6-97F0-B9767C596B07Q44089868-5AB15E12-CABE-4A82-AF8C-15B3B47FAA9AQ44570186-71E1B40D-AE85-40DC-8DCC-FC5C7F2BEB7BQ44805560-3DA2446A-8615-4C38-95BD-E90666233537Q46007457-8BABFDDD-DAF6-4892-BA42-E11F18A3EBCDQ46897374-40FB6A04-43A7-40A5-AD6F-C03E600485A9Q47687795-7F83CFEA-4B65-4DA2-9305-A1CC4385DA69Q48281408-D2DAF7C4-5193-4B5C-AEDC-E1C995B2493FQ48670206-3156FE3C-0984-4026-AF0B-3D920FD31B5F
P2860
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@en
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@nl
type
label
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@en
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@nl
prefLabel
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@en
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@nl
P2860
P1476
Sepsis induces diaphragm electron transport chain dysfunction and protein depletion.
@en
P2093
Gerald S Supinski
Leigh A Callahan
P2860
P304
P356
10.1164/RCCM.200410-1344OC
P407
P577
2005-06-30T00:00:00Z