Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects. - Wikidata - awgldk - TriplyDB

Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects.

Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects.

http://www.wikidata.org/entity/Q46604608

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Physiology of kidney renin Evidence that the vasodilator angiotensin-(1-7)-Mas axis plays an important role in erectile function Acute kidney injury in the rat causes cardiac remodelling and increases angiotensin-converting enzyme 2 expression Angiotensin-(1-7) and the g protein-coupled receptor MAS are key players in renal inflammation Angiotensin-(1-7) suppresses the number and function of the circulating fibrocytes by upregulating endothelial nitric oxide synthase expression.Autocrine and paracrine function of Angiotensin 1-7 in tissue repair during hypertension.Infusion of angiotensin-(1-7) reduces glomerulosclerosis through counteracting angiotensin II in experimental glomerulonephritis Effects of ACE2 inhibition in the post-myocardial infarction heart.Angiotensin converting enzyme (ACE) and ACE2 bind integrins and ACE2 regulates integrin signalling.The angiotensin-converting enzyme 2/angiogenesis-(1-7)/Mas axis confers cardiopulmonary protection against lung fibrosis and pulmonary hypertension.Discoidin domain receptor 2 germline gene deletion leads to altered heart structure and function in the mouse Cardiac-specific leptin receptor deletion exacerbates ischaemic heart failure in mice.Angiotensin-(1-7) infusion is associated with increased blood pressure and adverse cardiac remodelling in rats with subtotal nephrectomy.The novel Mas agonist, CGEN-856S, attenuates isoproterenol-induced cardiac remodeling and myocardial infarction injury in rats.MAS promoter regulation: a role for Sry and tyrosine nitration of the KRAB domain of ZNF274 as a feedback mechanism.Angiotensin-converting enzyme 2 activation protects against hypertension-induced cardiac fibrosis involving extracellular signal-regulated kinases.Targeting the ACE2-Ang-(1-7) pathway in cardiac fibroblasts to treat cardiac remodeling and heart failure Loss of ACE2 exacerbates murine renal ischemia-reperfusion injury.Characterization of the cardiac renin angiotensin system in oophorectomized and estrogen-replete mRen2.Lewis rats.ACE2 is augmented in dystrophic skeletal muscle and plays a role in decreasing associated fibrosis.Cardioprotective effects of telmisartan against heart failure in rats induced by experimental autoimmune myocarditis through the modulation of angiotensin-converting enzyme-2/angiotensin 1-7/mas receptor axis.ACE2-Ang (1-7) axis is induced in pressure overloaded rat model.Loss of prolyl carboxypeptidase in two-kidney, one-clip goldblatt hypertensive mice Angiotensin converting enzyme 2, Angiotensin-(1-7), and receptor MAS axis in the kidney.Beneficial effects of long-term administration of an oral formulation of Angiotensin-(1-7) in infarcted rats.Angiotensin-(1-7) abrogates mitogen-stimulated proliferation of cardiac fibroblasts.Angiotensin-(1-7) attenuates angiotensin II-induced cardiac remodeling associated with upregulation of dual-specificity phosphatase 1 Antagonism of angiotensin 1-7 prevents the therapeutic effects of recombinant human ACE2 International Union of Basic and Clinical Pharmacology. XCIX. Angiotensin Receptors: Interpreters of Pathophysiological Angiotensinergic Stimuli [corrected].Angiotensin-converting-enzyme 2 inhibits liver fibrosis in mice.Valsartan in the treatment of heart attack survivors Moderate cardiac-selective overexpression of angiotensin II type 2 receptor protects cardiac functions from ischaemic injury.ACE2, angiotensin-(1-7) and Mas receptor axis in inflammation and fibrosis.Role of the ACE2/Angiotensin 1-7 Axis of the Renin-Angiotensin System in Heart Failure Inhibition of angiotensin-converting enzyme 2 exacerbates cardiac hypertrophy and fibrosis in Ren-2 hypertensive rats.Angiotensin converting enzyme 2 (ACE2) activity in fetal calf serum: implications for cell culture research.New angiotensins.Distinct roles for angiotensin-converting enzyme 2 and carboxypeptidase A in the processing of angiotensins within the murine heart Portal pressure responses and angiotensin peptide production in rat liver are determined by relative activity of ACE and ACE2.Mitigation of radiation induced pulmonary vascular injury by delayed treatment with captopril.

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P2860

Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects.

http://www.wikidata.org/entity/Q46604608

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2005 nî lūn-bûn

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2005年学术文章

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2005年學術文章

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2005年學術文章

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Angiotensin-(1-7) binds to spe ...... rotic and antitrophic effects.

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AJPHEART.00317.2005

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American Journal of Physiology - Heart and Circulatory Physiology

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Angiotensin-(1-7) binds to spe ...... rotic and antitrophic effects.

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Barry H Greenberg

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Cristina Moore

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Devorah Gurantz

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Jason X-J Yuan

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Michikado Iwata

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Randy T Cowling

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Shen Zhang

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P2860

A human homolog of angiotensin-converting enzyme. Cloning and functional expression as a captopril-insensitive carboxypeptidase

Angiotensin-(1-7) is an endogenous ligand for the G protein-coupled receptor Mas

Hydrolysis of biological peptides by human angiotensin-converting enzyme-related carboxypeptidase

Angiotensin-converting enzyme 2 is an essential regulator of heart function

The cardiac renin-angiotensin system: conceptual, or a regulator of cardiac function?

Gene expression in fibroblasts and fibrosis: involvement in cardiac hypertrophy.

Angiotensin-(1-7): an update.

Characterization of angiotensin II receptors in cultured adult rat cardiac fibroblasts. Coupling to signaling systems and gene expression.

Compartmentalization of angiotensin II generation in the dog heart. Evidence for independent mechanisms in intravascular and interstitial spaces.

Tumor necrosis factor-alpha upregulates angiotensin II type 1 receptors on cardiac fibroblasts.

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10.1152/AJPHEART.00317.2005

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6

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289

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16024575

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