pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
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Investigational antiarrhythmic agents: promising drugs in early clinical development.The Impact of Heterozygous KCNK3 Mutations Associated With Pulmonary Arterial Hypertension on Channel Function and Pharmacological Recovery.Termination of Vernakalant-Resistant Atrial Fibrillation by Inhibition of Small-Conductance Ca2+-Activated K+ Channels in Pigs.
P2860
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
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2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
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2016年學術文章
@yue
2016年學術文章
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name
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@en
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@nl
type
label
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@en
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@nl
prefLabel
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@en
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@nl
P2093
P2860
P50
P1433
P1476
pH-dependent inhibition of K₂P3.1 prolongs atrial refractoriness in whole hearts.
@en
P2093
Emilie Gregers
Jonas G Diness
Lea Abildgaard
Morten Grunnet
Nicole Schmitt
Ulrik S Sørensen
P2860
P2888
P304
P356
10.1007/S00424-015-1779-0
P50
P577
2016-01-05T00:00:00Z