Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
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Siglecs and immune regulationCarbohydrate Recognition Properties of Human Ficolins: GLYCAN ARRAY SCREENING REVEALS THE SIALIC ACID BINDING SPECIFICITY OF M-FICOLINPorcine sialoadhesin (CD169/Siglec-1) is an endocytic receptor that allows targeted delivery of toxins and antigens to macrophages9- O-acetylated sialic acids enhance entry of virulent Leishmania donovani promastigotes into macrophages.Role of macrophage sialoadhesin in host defense against the sialylated pathogen group B Streptococcus.The interplay between Siglecs and sialylated pathogensCulture of mouse peritoneal macrophages with mouse serum induces lipid bodies that associate with the parasitophorous vacuole and decrease their microbicidal capacity against Toxoplasma gondii.Interferon-inducible mechanism of dendritic cell-mediated HIV-1 dissemination is dependent on Siglec-1/CD169.Distinct endocytic mechanisms of CD22 (Siglec-2) and Siglec-F reflect roles in cell signaling and innate immunity.Sialoadhesin promotes rapid proinflammatory and type I IFN responses to a sialylated pathogen, Campylobacter jejuni.Induction of Siglec-1 by Endotoxin Tolerance Suppresses the Innate Immune Response by Promoting TGF-β1 Production.Acute heart inflammation: ultrastructural and functional aspects of macrophages elicited by Trypanosoma cruzi infectionBasic and clinical immunology of SiglecsSiglecs as targets for therapy in immune-cell-mediated disease.Sialic acids siglec interaction: a unique strategy to circumvent innate immune response by pathogens.The chemistry and biology of trypanosomal trans-sialidases: virulence factors in Chagas disease and sleeping sickness.Innate immune functions of macrophage subpopulations in the spleen.Sialoadhesin in recognition of self and non-self.Sialoadhesin - a macrophage-restricted marker of immunoregulation and inflammation.How do pathogens drive the evolution of paired receptors?F4/80 as a Major Macrophage Marker: The Case of the Peritoneum and Spleen.Invasion of Trypanosoma cruzi into host cells is impaired by N-propionylmannosamine and other N-acylmannosamines.Chronic Brucella Infection Induces Selective and Persistent Interferon Gamma-Dependent Alterations of Marginal Zone Macrophages in the Spleen.Targeting to porcine sialoadhesin receptor improves antigen presentation to T cells.CD169 Macrophages Capture and Dendritic Cells Instruct: The Interplay of the Gatekeeper and the General of the Immune System
P2860
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P2860
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
description
2005 nî lūn-bûn
@nan
2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
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2005年學術文章
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name
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
@en
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
@nl
type
label
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
@en
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
@nl
prefLabel
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
@en
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages.
@nl
P2093
P1476
Increased association of Trypanosoma cruzi with sialoadhesin positive mice macrophages
@en
P2093
Alexandre R Silva
Caroliny S S Lobato
Diego V Medina
Sergio H Seabra
Verônica G Monteiro
Wanderley de Souza
P2860
P2888
P304
P356
10.1007/S00436-005-1460-1
P577
2005-09-07T00:00:00Z