Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass. - Wikidata - awgldk - TriplyDB

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

http://www.wikidata.org/entity/Q46693499

about

Effects of biological sex on the pathophysiology of the heart Effects of sex steroids on bones and muscles: Similarities, parallels, and putative interactions in health and disease Estrogen regulates estrogen receptors and antioxidant gene expression in mouse skeletal muscle.Estrogen replacement and skeletal muscle: mechanisms and population health.Musculoskeletal response to whole-body vibration during fracture healing in intact and ovariectomized rats Benefits of estrogen replacement for skeletal muscle mass and function in post-menopausal females: evidence from human and animal studies Influence of ovarian hormones on strength loss in healthy and dystrophic female mice.Lipolytic signaling in response to acute exercise is altered in female mice following ovariectomy Resistance training associated with the administration of anabolic-androgenic steroids improves insulin sensitivity in ovariectomized rats.Diminished satellite cells and elevated adipogenic gene expression in muscle as caused by ovariectomy are averted by low-magnitude mechanical signals.Estradiol modulates myosin regulatory light chain phosphorylation and contractility in skeletal muscle of female mice.Ectopic lipid deposition and the metabolic profile of skeletal muscle in ovariectomized mice.Sexual dimorphism in skeletal muscle protein turnover The role of androgens and estrogens on healthy aging and longevity.Identification of a vibration regime favorable for bone healing and muscle in estrogen-deficient rats FKBP12 deficiency reduces strength deficits after eccentric contraction-induced muscle injury.Using a novel coculture model to dissect the role of intramuscular lipid load on skeletal muscle insulin responsiveness under reduced estrogen conditions.Estrogen receptor-alpha and -beta and aromatase knockout effects on lower limb muscle mass and contractile function in female mice.Acute resistance exercise reduces increased gene expression in muscle atrophy of ovariectomised arthritic rats.Menopause, estrogens and frailty.Female hormones: do they influence muscle and tendon protein metabolism?8-Prenylnaringenin promotes recovery from immobilization-induced disuse muscle atrophy through activation of the Akt phosphorylation pathway in mice.Chronic high fat feeding attenuates load-induced hypertrophy in mice.Wheel running prevents the accumulation of monounsaturated fatty acids in the liver of ovariectomized mice by attenuating changes in SCD-1 content.Point:Counterpoint: Estrogen and sex do/do not influence post-exercise indexes of muscle damage, inflammation, and repair.Skeletal muscle and bone: effect of sex steroids and aging.Estrogen influences satellite cell activation and proliferation following downhill running in rats.A functional insulin-like growth factor receptor is not necessary for load-induced skeletal muscle hypertrophy.Estrogen status and skeletal muscle recovery from disuse atrophy.The effects of castration and hormone replacement on the cross-sectional area of pubococcygeus muscle fibers in the female rat.A rehabilitation exercise program to remediate skeletal muscle atrophy in an estrogen-deficient organism may be ineffective.Estradiol replacement reverses ovariectomy-induced muscle contractile and myosin dysfunction in mature female mice.Ovarian hormone status and skeletal muscle inflammation during recovery from disuse in rats.Involvement of AMPK in regulating slow-twitch muscle atrophy during hindlimb unloading in mice.Oestrogen-dependent satellite cell activation and proliferation following a running exercise occurs via the PI3K signalling pathway and not IGF-1.The role of the estrogen receptor in skeletal muscle mass homeostasis and regeneration.Muscle and Bone Health in Postmenopausal Women: Role of Protein and Vitamin D Supplementation Combined with Exercise Training Gonadotropins Are Related to Lean Mass in Healthy Postmenopausal Women

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P2860

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

http://www.wikidata.org/entity/Q46693499

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@en

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@nl

type

label

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@en

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@nl

prefLabel

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@en

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@nl

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JAPPLPHYSIOL.00869.2005

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Journal of Applied Physiology

P1476

Ovariectomy prevents the recovery of atrophied gastrocnemius skeletal muscle mass.

@en

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Andrea M Foley

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Espen E Spangenburg

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Marybeth Brown

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Mitchell Sitnick

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P2860

Exercise and gene expression: physiological regulation of the human genome through physical activity

Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

Signalling pathways that mediate skeletal muscle hypertrophy and atrophy

Phosphorylation of mammalian target of rapamycin (mTOR) at Ser-2448 is mediated by p70S6 kinase

Identification of S6 kinase 1 as a novel mammalian target of rapamycin (mTOR)-phosphorylating kinase

Contraction-mediated mTOR, p70S6k, and ERK1/2 phosphorylation in aged skeletal muscle

Mammalian cell size is controlled by mTOR and its downstream targets S6K1 and 4EBP1/eIF4E

Control of Ser2448 phosphorylation in the mammalian target of rapamycin by insulin and skeletal muscle load.

Atrophy responses to muscle inactivity. II. Molecular markers of protein deficits.

Intracellular signaling pathways: nongenomic actions of estrogens and ligand-independent activation of estrogen receptors.

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