Dynamics of metalloproteinase-2 and -9, TGF-beta, and uPA activities during normoxic vs. hyperoxic alveolarization.
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The Extracellular Matrix in Bronchopulmonary Dysplasia: Target and SourceMatrix metalloproteinase gene polymorphisms and bronchopulmonary dysplasia: identification of MMP16 as a new player in lung developmentTransforming growth factor-beta modulates the expression of nitric oxide signaling enzymes in the injured developing lung and in vascular smooth muscle cells.A potential role of the JNK pathway in hyperoxia-induced cell death, myofibroblast transdifferentiation and TGF-β1-mediated injury in the developing murine lung.Differential expression of MMP-2 and -9 and their inhibitors in fetal lung cells exposed to mechanical stretch: regulation by IL-10.The early responses of VEGF and its receptors during acute lung injury: implication of VEGF in alveolar epithelial cell survival.Hyperoxia enhances VEGF release from A549 cells via post-transcriptional processes.Control mechanisms of lung alveolar development and their disorders in bronchopulmonary dysplasia.Similarities and dissimilarities of branching and septation during lung development.Matrix metalloproteinases in lung: multiple, multifarious, and multifaceted.Role of matrix metalloprotease-9 in hyperoxic injury in developing lung.Maturational differences in lung NF-kappaB activation and their role in tolerance to hyperoxiaThymulin evokes IL-6-C/EBPbeta regenerative repair and TNF-alpha silencing during endotoxin exposure in fetal lung explants.A role for matrix metalloproteinase 9 in IFNγ-mediated injury in developing lungs: relevance to bronchopulmonary dysplasia.Hyperoxia modulates TGF-beta/BMP signaling in a mouse model of bronchopulmonary dysplasia.Gene expression profile in newborn rat lungs after two days of recovery of mechanical ventilation.TGF-beta signaling promotes survival and repair in rat alveolar epithelial type 2 cells during recovery after hyperoxic injury.Increased expression of inflammatory genes in the neonatal mouse brain after hyperoxic reoxygenation.TGF-beta-neutralizing antibodies improve pulmonary alveologenesis and vasculogenesis in the injured newborn lung.Conditional overexpression of bioactive transforming growth factor-beta1 in neonatal mouse lung: a new model for bronchopulmonary dysplasia?Alteration of TGF-β-ALK-Smad signaling in hyperoxia-induced bronchopulmonary dysplasia model of newborn rats.Activin A contributes to the development of hyperoxia-induced lung injury in neonatal mice.
P2860
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P2860
Dynamics of metalloproteinase-2 and -9, TGF-beta, and uPA activities during normoxic vs. hyperoxic alveolarization.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@en
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@nl
type
label
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@en
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@nl
prefLabel
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@en
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@nl
P2860
P1476
Dynamics of metalloproteinase- ...... vs. hyperoxic alveolarization.
@en
P2093
Warburton D
P2860
P304
P356
10.1152/AJPLUNG.00415.2001
P577
2002-10-01T00:00:00Z