Combination of proteasome and class I HDAC inhibitors induces apoptosis of NPC cells through an HDAC6-independent ER stress-induced mechanism.
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Carfilzomib combined with suberanilohydroxamic acid (SAHA) synergistically promotes endoplasmic reticulum stress in non-small cell lung cancer cell lines.Induction of MAPK- and ROS-dependent autophagy and apoptosis in gastric carcinoma by combination of romidepsin and bortezomibHistone deacetylase inhibitor panobinostat induces calcineurin degradation in multiple myeloma.HDAC Inhibitors and RECK Modulate Endoplasmic Reticulum Stress in Tumor Cells.Caffeic acid phenethyl ester enhances TRAIL-mediated apoptosis via CHOP-induced death receptor 5 upregulation in hepatocarcinoma Hep3B cells.Low-dose spiruchostatin-B, a potent histone deacetylase inhibitor enhances radiation-induced apoptosis in human lymphoma U937 cells via modulation of redox signaling.Investigational drugs for head and neck cancer.Inhibition of class I histone deacetylases by romidepsin potently induces Epstein-Barr virus lytic cycle and mediates enhanced cell death with ganciclovir.HDACs and HDAC Inhibitors in Cancer Development and Therapy.Combination of SAHA and bortezomib up-regulates CDKN2A and CDKN1A and induces apoptosis of Epstein-Barr virus-positive Wp-restricted Burkitt lymphoma and lymphoblastoid cell lines.Influence of Intensity-Modulated Radiation Therapy on the Life Quality of Patients with Nasopharyngeal Carcinoma.Analysis of the interplay between all-trans retinoic acid and histone deacetylase inhibitors in leukemic cells.The histone deacetylase inhibitor romidepsin synergizes with lenalidomide and enhances tumor cell death in T-cell lymphoma cell lines.Therapeutic Strategies against Epstein-Barr Virus-Associated Cancers Using Proteasome Inhibitors.Combination Therapy With Histone Deacetylase Inhibitors (HDACi) for the Treatment of Cancer: Achieving the Full Therapeutic Potential of HDACi.Selective Histone Deacetylase 6 Inhibitor 23BB Alleviated Rhabdomyolysis-Induced Acute Kidney Injury by Regulating Endoplasmic Reticulum Stress and Apoptosis.Counteracting survival functions of EBNA3C in Epstein-Barr virus (EBV)-driven lymphoproliferative diseases by combination of SAHA and bortezomib.
P2860
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P2860
Combination of proteasome and class I HDAC inhibitors induces apoptosis of NPC cells through an HDAC6-independent ER stress-induced mechanism.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh
2014年學術文章
@zh-hant
name
Combination of proteasome and ...... t ER stress-induced mechanism.
@en
Combination of proteasome and ...... t ER stress-induced mechanism.
@nl
type
label
Combination of proteasome and ...... t ER stress-induced mechanism.
@en
Combination of proteasome and ...... t ER stress-induced mechanism.
@nl
prefLabel
Combination of proteasome and ...... t ER stress-induced mechanism.
@en
Combination of proteasome and ...... t ER stress-induced mechanism.
@nl
P2860
P356
P1476
Combination of proteasome and ...... t ER stress-induced mechanism.
@en
P2093
Kwai Fung Hui
P2860
P304
P356
10.1002/IJC.28924
P577
2014-05-05T00:00:00Z