In vivo inhibition of nitric oxide synthase impairs upregulation of contractile protein mRNA in overloaded plantaris muscle.
about
PGC-1alpha regulation by exercise training and its influences on muscle function and insulin sensitivity.Exercise training modulates the nitric oxide synthase profile in skeletal muscle from old rats.Exercise-induced skeletal muscle remodeling and metabolic adaptation: redox signaling and role of autophagy.Impaired adaptive response to mechanical overloading in dystrophic skeletal muscleArginine protects muscle cells from wasting in vitro in an mTORC1-dependent and NO-independent manner.Loss of nNOS inhibits compensatory muscle hypertrophy and exacerbates inflammation and eccentric contraction-induced damage in mdx mice.L-Citrulline Protects Skeletal Muscle Cells from Cachectic Stimuli through an iNOS-Dependent Mechanism.Effects of Nitric Oxide Synthase Inhibition on Fiber-Type Composition, Mitochondrial Biogenesis, and SIRT1 Expression in Rat Skeletal MuscleSkeletal muscle function during exercise-fine-tuning of diverse subsystems by nitric oxideAge-related loss of nitric oxide synthase in skeletal muscle causes reductions in calpain S-nitrosylation that increase myofibril degradation and sarcopenia.L-Arginine as a potential ergogenic aid in healthy subjects.Potential mechanisms for a role of metabolic stress in hypertrophic adaptations to resistance training.Characterization and regulation of mechanical loading-induced compensatory muscle hypertrophy.No-dependent signaling pathways in unloaded skeletal muscle.Redox control of skeletal muscle atrophy.Activation of calcium signaling through Trpv1 by nNOS and peroxynitrite as a key trigger of skeletal muscle hypertrophy.Nitric oxide facilitates NFAT-dependent transcription in mouse myotubes.Basal, but not overload-induced, myonuclear addition is attenuated by NG-nitro-L-arginine methyl ester (L-NAME) administration.Nitric oxide generated by muscle corrects defects in hippocampal neurogenesis and neural differentiation caused by muscular dystrophy.Citrulline does not prevent skeletal muscle wasting or weakness in limb-casted mice.Glucose deprivation promotes activation of mTOR signaling pathway and protein synthesis in rat skeletal muscle cells.Nitric oxide synthase inhibition delays low-frequency stimulation-induced satellite cell activation in rat fast-twitch muscle.Nitric oxide synthase inhibition prevents activity-induced calcineurin-NFATc1 signalling and fast-to-slow skeletal muscle fibre type conversions.
P2860
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P2860
In vivo inhibition of nitric oxide synthase impairs upregulation of contractile protein mRNA in overloaded plantaris muscle.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@en
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@nl
type
label
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@en
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@nl
prefLabel
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@en
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@nl
P2093
P2860
P1476
In vivo inhibition of nitric o ...... n overloaded plantaris muscle.
@en
P2093
David S Criswell
Jeff E Sellman
Jenna L Betters
Joshua T Selsby
Keith C DeRuisseau
Quinlyn A Soltow
Vitor A Lira
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00936.2005
P407
P577
2005-09-15T00:00:00Z