High glucose-induced human umbilical vein endothelial cell hyperpermeability is dependent on protein kinase C activation and independent of the Ca2+-nitric oxide signalling pathway.

High glucose-induced human umbilical vein endothelial cell hyperpermeability is dependent on protein kinase C activation and independent of the Ca2+-nitric oxide signalling pathway.

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http://www.wikidata.org/entity/Q46712828
Diabetes alters intracellular calcium transients in cardiac endothelial cells.Involvement of RhoA/ROCK1 signaling pathway in hyperglycemia-induced microvascular endothelial dysfunction in diabetic retinopathyCitrate treatment reduces endothelial death and inflammation under hyperglycaemic conditions.Hyperglycemia and endothelial dysfunction in atherosclerosis: lessons from type 1 diabetes.Increased susceptibility to amyloid-β toxicity in rat brain microvascular endothelial cells under hyperglycemic conditions.Resveratrol ameliorates high-glucose-induced hyperpermeability mediated by caveolae via VEGF/KDR pathwayDifferential function and regulation of orphan nuclear receptor TR3 isoforms in endothelial cells.Ascorbate reverses high glucose- and RAGE-induced leak of the endothelial permeability barrierAcute and sustained actions of hyperglycaemia on endothelial and glomerular barrier permeability.Endothelial barrier protection by FTY720 under hyperglycemic condition: involvement of focal adhesion kinase, small GTPases, and adherens junction proteins.Disruption of endothelial adherens junctions by high glucose is mediated by protein kinase C-β-dependent vascular endothelial cadherin tyrosine phosphorylation.Disruption of endothelial adherens junctions by high glucose is mediated by protein kinase C-β–dependent vascular endothelial cadherin tyrosine phosphorylation.Rhodiola crenulata Attenuates High Glucose Induced Endothelial Dysfunction in Human Umbilical Vein Endothelial Cells.The locally activated renin-angiotensin system is involved in albumin permeability in glomerular endothelial cells under high glucose conditions.Aquaporin-1 channel function is positively regulated by protein kinase C.
P2860

High glucose-induced human umbilical vein endothelial cell hyperpermeability is dependent on protein kinase C activation and independent of the Ca2+-nitric oxide signalling pathway.

Item
http://www.wikidata.org/entity/Q46712828
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
High glucose-induced human umb ...... tric oxide signalling pathway.
@en
High glucose-induced human umb ...... tric oxide signalling pathway.
@nl
type
label
High glucose-induced human umb ...... tric oxide signalling pathway.
@en
High glucose-induced human umb ...... tric oxide signalling pathway.
@nl
prefLabel
High glucose-induced human umb ...... tric oxide signalling pathway.
@en
High glucose-induced human umb ...... tric oxide signalling pathway.
@nl
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P2860
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P698
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High glucose-induced human umb ...... tric oxide signalling pathway.
@en
P2093
P2860
P304
P31
P356
10.1111/J.1440-1681.2005.04266.X
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P478
P577
P698