Selenium prevents diabetes-induced alterations in [Zn2+]i and metallothionein level of rat heart via restoration of cell redox cycle.
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Myosin Binding Protein C Positioned to Play a Key Role in Regulation of Muscle Contraction: Structure and Interactions of Domain C1ß-blocker timolol prevents arrhythmogenic Ca²⁺ release and normalizes Ca²⁺ and Zn²⁺ dyshomeostasis in hyperglycemic rat heart.Vimentin filament organization and stress sensing depend on its single cysteine residue and zinc binding.Intracellular Zinc Modulates Cardiac Ryanodine Receptor-mediated Calcium Release.Cellular zinc and redox buffering capacity of metallothionein/thionein in health and diseaseQuantitative imaging of mitochondrial and cytosolic free zinc levels in an in vitro model of ischemia/reperfusion.Zinc biochemistry: from a single zinc enzyme to a key element of life.Oxidant balance in brain of rats receiving different compounds of selenium.Enhancement of cellular antioxidant-defence preserves diastolic dysfunction via regulation of both diastolic Zn2+ and Ca2+ and prevention of RyR2-leak in hyperglycemic cardiomyocytes.Resveratrol and diabetic cardiac function: focus on recent in vitro and in vivo studies.Zinc homeostasis in the metabolic syndrome and diabetes.Dysregulated Zn2+ homeostasis impairs cardiac type-2 ryanodine receptor and mitsugumin 23 functions, leading to sarcoplasmic reticulum Ca2+ leakage.Heart selenoproteins status of metabolic syndrome-exposed pups: A potential target for attenuating cardiac damage.Zinc in Pancreatic Islet Biology, Insulin Sensitivity, and Diabetes.Zinc controls RyR2 activity during excitation-contraction coupling.Different redox states of metallothionein/thionein in biological tissue.Immuno-spin trapping detection of antioxidant/pro-oxidant properties of zinc or selenium on DNA and protein radical formation via hydrogen peroxide.Intracellular Zn(2+) Increase in Cardiomyocytes Induces both Electrical and Mechanical Dysfunction in Heart via Endogenous Generation of Reactive Nitrogen Species.Protective roles of selenium on nitric oxide-mediated apoptosis of immune organs induced by cadmium in chickens.Zinc modulation of basal and β-adrenergically stimulated L-type Ca2+ current in rat ventricular cardiomyocytes: consequences in cardiac diseases.Protective action of doxycycline against diabetic cardiomyopathy in rats.Impact of Labile Zinc on Heart Function: From Physiology to Pathophysiology.Selenoproteins and cardiovascular stress.The Effect of Selenium on the Cd-Induced Apoptosis via NO-Mediated Mitochondrial Apoptosis Pathway in Chicken Liver.
P2860
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P2860
Selenium prevents diabetes-induced alterations in [Zn2+]i and metallothionein level of rat heart via restoration of cell redox cycle.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@en
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@nl
type
label
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@en
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@nl
prefLabel
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@en
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@nl
P2860
P1476
Selenium prevents diabetes-ind ...... storation of cell redox cycle.
@en
P2093
Belma Turan
Murat Ayaz
P2860
P304
P356
10.1152/AJPHEART.00754.2005
P577
2005-10-07T00:00:00Z