ANG II provokes acute trafficking of distal tubule Na+-Cl(-) cotransporter to apical membrane. - Wikidata - awgldk - TriplyDB

ANG II provokes acute trafficking of distal tubule Na+-Cl(-) cotransporter to apical membrane.

ANG II provokes acute trafficking of distal tubule Na+-Cl(-) cotransporter to apical membrane.

http://www.wikidata.org/entity/Q46774246

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Molecular physiology of the thiazide-sensitive sodium-chloride cotransporter The thiazide-sensitive Na+-Cl- cotransporter: molecular biology, functional properties, and regulation by WNKs Angiotensin II signaling increases activity of the renal Na-Cl cotransporter through a WNK4-SPAK-dependent pathway Interacting influence of diuretics and diet on BK channel-regulated K homeostasis Integrated control of Na transport along the nephron.Renal NCC is unchanged in the midpregnant rat and decreased in the late pregnant rat despite avid renal Na+ retention Effects of dietary salt on renal Na+ transporter subcellular distribution, abundance, and phosphorylation status A SPAK isoform switch modulates renal salt transport and blood pressure Scaffold protein connector enhancer of kinase suppressor of Ras isoform 3 (CNK3) coordinates assembly of a multiprotein epithelial sodium channel (ENaC)-regulatory complex Angiotensin II stimulates trafficking of NHE3, NaPi2, and associated proteins into the proximal tubule microvilli.Sympathetic stimulation of thiazide-sensitive sodium chloride cotransport in the generation of salt-sensitive hypertension.Mechanisms of proximal tubule sodium transport regulation that link extracellular fluid volume and blood pressure.Acute insulin stimulation induces phosphorylation of the Na-Cl cotransporter in cultured distal mpkDCT cells and mouse kidney.RasGRP1 stimulation enhances ubiquitination and endocytosis of the sodium-chloride cotransporter Aldosterone acutely stimulates NCC activity via a SPAK-mediated pathway.WNK4 is the major WNK positively regulating NCC in the mouse kidney Gene expression profiles linked to AT1 angiotensin receptors in the kidney.The WNKs: atypical protein kinases with pleiotropic actions.Distal convoluted tubule Phorbol ester stimulation of RasGRP1 regulates the sodium-chloride cotransporter by a PKC-independent pathway.Effect of volume expansion with hypertonic- and isotonic saline and isotonic glucose on sodium and water transport in the principal cells in the kidney Conservation of Na+ vs. K+ by the rat cortical collecting duct.Nedd4-2 modulates renal Na+-Cl- cotransporter via the aldosterone-SGK1-Nedd4-2 pathway.Mechanisms of angiotensin II stimulation of NCC are time-dependent in mDCT15 cells.Hypertrophy in the Distal Convoluted Tubule of an 11β-Hydroxysteroid Dehydrogenase Type 2 Knockout Model.Potassium excretion during antinatriuresis: perspective from a distal nephron model.Activation of the renal Na+:Cl- cotransporter by angiotensin II is a WNK4-dependent process.Short-term nonpressor angiotensin II infusion stimulates sodium transporters in proximal tubule and distal nephron.Regulation of the renal Na+-Cl- cotransporter by phosphorylation and ubiquitylation.Generation and analysis of knock-in mice carrying pseudohypoaldosteronism type II-causing mutations in the cullin 3 gene.Direct activation of ENaC by angiotensin II: recent advances and new insights.Evolving concepts on regulation and function of renin in distal nephron.Reduced ENaC activity and blood pressure in mice with genetic knockout of the insulin receptor in the renal collecting duct.SPAK differentially mediates vasopressin effects on sodium cotransporters.Effects of ACE inhibition and ANG II stimulation on renal Na-Cl cotransporter distribution, phosphorylation, and membrane complex properties Norepinephrine-evoked salt-sensitive hypertension requires impaired renal sodium chloride cotransporter activity in Sprague-Dawley rats.Potassium and Its Discontents: New Insight, New Treatments Multigene kinase network, kidney transport, and salt in essential hypertension.Maintaining Balance Under Pressure: Integrated Regulation of Renal Transporters During Hypertension Acute hypertension provokes acute trafficking of distal tubule Na-Cl cotransporter (NCC) to subapical cytoplasmic vesicles

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P2860

ANG II provokes acute trafficking of distal tubule Na+-Cl(-) cotransporter to apical membrane.

http://www.wikidata.org/entity/Q46774246

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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2007年學術文章

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name

ANG II provokes acute trafficking of distal tubule Na+-Cl

@nl

ANG II provokes acute traffick ...... ransporter to apical membrane.

@en

type

label

ANG II provokes acute trafficking of distal tubule Na+-Cl

@nl

ANG II provokes acute traffick ...... ransporter to apical membrane.

@en

prefLabel

ANG II provokes acute trafficking of distal tubule Na+-Cl

@nl

ANG II provokes acute traffick ...... ransporter to apical membrane.

@en

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AJPRENAL.00064.2007

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American Journal of Physiology - Renal Physiology

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ANG II provokes acute traffick ...... ransporter to apical membrane.

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Alicia A McDonough

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Anne D M Riquier

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Arvid B Maunsbach

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Kaarina Pihakaski-Maunsbach

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Monica B Sandberg

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P2860

Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Functional expression of mutations in the human NaCl cotransporter: evidence for impaired routing mechanisms in Gitelman's syndrome

The renal thiazide-sensitive Na-Cl cotransporter as mediator of the aldosterone-escape phenomenon

Redistribution of distal tubule Na+-Cl- cotransporter (NCC) in response to a high-salt diet

Distribution of angiotensin AT1 and AT2 receptor subtypes in the rat kidney

The serum- and glucocorticoid-induced kinase is a physiological mediator of aldosterone action

WNK kinases regulate thiazide-sensitive Na-Cl cotransport

Effects of ACE inhibition on proximal tubule sodium transport.

Mechanisms of WNK1 and WNK4 interaction in the regulation of thiazide-sensitive NaCl cotransport

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