Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
about
Enhanced nitrosative stress during Trypanosoma cruzi infection causes nitrotyrosine modification of host proteins: implications in Chagas' disease.Protein targets of tyrosine nitration in sunflower (Helianthus annuus L.) hypocotyls.NADPH phagocyte oxidase knockout mice control Trypanosoma cruzi proliferation, but develop circulatory collapse and succumb to infection.Gamma delta T cells recognize haptens and mount a hapten-specific response.P47phox-/- mice are compromised in expansion and activation of CD8+ T cells and susceptible to Trypanosoma cruzi infection.Nitrative thioredoxin inactivation as a cause of enhanced myocardial ischemia/reperfusion injury in the aging heart.Increased cytokine and nitric oxide levels in serum of dogs experimentally infected with Rangelia vitaliiTrypanosoma cruzi antioxidant enzymes as virulence factors in Chagas disease.Increased myeloperoxidase activity and protein nitration are indicators of inflammation in patients with Chagas' disease.
P2860
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P2860
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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name
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@en
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@nl
type
label
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@en
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@nl
prefLabel
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@en
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@nl
P2093
P2860
P1476
Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice.
@en
P2093
P2860
P304
P356
10.1590/S0100-879X2005001200011
P407
P577
2005-11-09T00:00:00Z